氧化损伤；
CONCLUSION CTWA may delay aging, which may be related to antagonizing free radical injury and enhancing immunity of aging mice.
结论CT WA能拮抗自由基损伤，增强衰老小鼠心和脑组织端粒酶活性和机体免疫功能，这些可能与CT WA的抗衰老作用有关。
Conclusion: the calcium overload and free radical injury were correlated with brain ischemia reperfusion injury in the aged rats.
结论：老龄大鼠脑缺血再灌注脑组织损伤与钙超载和自由基损伤有关。
Objective: To observe the dynamic changes of free radical of mice with immune liver injury and the preventive effects of Liganshu Granules on the free radical injury in mice with immune liver injury.
目的：观察免疫性肝损伤小鼠自由基的动态变化以及利肝舒冲剂对免疫性肝损伤小鼠自由基损伤的预防作用。
Hyperglycemia has been shown to potentiate ischemic injury of the spinal cord by quenching vasodilators and potentiating tissue acidosis and free radical production.
目的：已有事实表明高血糖症可通过减弱血管扩张，提高组织酸度，释放刺激性产物导致脊髓的缺血损伤。
Phenytoin sodium may protect brain tissues from cerebral ischemic injury by inhibiting formation of free radical, anti-lipid peroxidation and improving regional cerebral blood flow.
苯妥英钠对缺血性脑损伤的神经保护作用与其能抑制自由基形成，减轻自由基介导的毒性，抗脂质过氧化反应以及扩张血管，增加局部脑血流量有关。
Conclusion Insulin has a protective effect against myocardial ischemia and reperfusion injury in rat, the protective mechanism involves antiapoptotic and antioxide free radical effect of insulin.
结论胰岛素对大鼠再灌注心肌损伤具有保护作用，其保护机制可能与抑制细胞凋亡及抗氧自由基作用有关。
Conclusions the use of anisodamine before reperfusion could reduce the production of oxygen derived free radical, therefore alleviate the injury of grafted liver caused by ischemia reperfusion.
结论再灌流前应用山茛菪碱能减少再灌流后氧自由基的产生，可减轻缺血—再灌流对移植肝脏的损伤。
Objective: To research Huanongjiu santi-free radical effects on chronic immuno-hepatic injury of mice.
目的：研究化脓灸对免疫性慢性肝损伤小鼠抗自由基损伤的作用。
Aim: To investigate the effect of TRH on free radical reactions in brain tissues after acute experimental brain injury.
目的：探讨促甲状腺素释放激素(TRH)对急性实验性颅脑损伤后脑组织自由基反应的影响。
In recent years more and more research results have showed that free radical-caused injury has a very important place in etiology of ischemic cerebrovascular disease.
近年来，愈来愈多的研究结果表明，自由基损伤在缺血性脑血管病的病原学方面占有很重要的位置。
CONCLUSION Icariin has protective effect on rat cerebral mitochondria injury induced by oxygen free radical.
结论淫羊藿苷对氧自由基损伤的大鼠脑线粒体呼吸链具有保护作用。
The studies of free radical during exercise on effects of the mechanisms of cell adaptive regulation and exercise-induced fatigue and the causality of sports injury are still no obvious development.
运动性内源自由基与细胞适应性调节机制、运动损伤的因果关系、疲劳发生的机制等方面研究目前仍无突破性进展。
The results showed that the anti free radical treatment could be used to alleviate the secondary tissue injury after fracture and surgical trauma.
结果表明抗自由基治疗可作为骨折及手术创伤后减轻组织继发性损伤的辅助措施。
Oxygen free radical may play important roles in myocardial ischemia-reperfusion injury after CPB.
氧自由基在CPB后心肌缺血再灌注损伤中起重要作用。
Aim: To investigate the effects of indomethacin (im) on free radical reactions in brain tissues of rabbits with acute experimental brain injury.
目的：探讨消炎痛对急性实验性脑外伤后兔脑组织氧自由基反应的影响。
Objective To study the changes of free radical in blood and spinal cord after spinal cord injury (SCI)and the elimination of free radical with Salvia Miltiorrhiza.
目的了解实验性脊髓损伤后血液和脊髓组织中自由基的变化及丹参对自由基的清除作用。
Objective It is discussed that the effect of tetrandrine to the eye injury caused by free radical.
目的探讨汉防己甲素对自由基眼损伤的影响。
Objective To study the change principles of the oxygen free radical on the sub-cellular level in the process of ischemia-reperfusion injury.
目的亚细胞水平上探索肝脏缺血再灌注损伤中氧自由基的变化规律。
CONCLUSION Hyperoxic CPB leads to direct myocardial and cerebral injury induced by oxygen free radical.
结论高氧分压体外循环再氧合可导致心肌和脑组织氧自由基介导的再氧合损伤；
Conclusion: The results demonstrate that the free radical-injury has an important effect on ARF rabbits subjected to glycerinum.
结论：自由基损伤在甘油致家兔ARF过程中具有重要作用。
Objective To study the role of leukocyte in generating oxygen free radical in myocardial ischemia and reperfusion injury by leukocyte-depletion technique.
目的采用在体犬心肌缺血再灌注动物实验模型，通过去白细胞技术，研究白细胞在缺血再灌注损伤心肌中产生氧自由基的作用。
Then, the free radical from the alcohol metabolism is one of the important factors that cause lipid peroxidation and alcoholic liver injury.
而酒精代谢过程中所产生的自由基，其引起的脂质过氧化反应是导致酒精性肝损伤的重要因素之一。
Free radical damnification might be one of the liver injury mechanism of peppermint oil.
自由基损伤可能是其肝毒性机制之一。
Free radical damnification might be one of the liver injury mechanism of peppermint oil.
自由基损伤可能是其肝毒性机制之一。
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