Prof. Jia: Fibrogenesis is not a single disease.
贾继东教授：纤维化不是一个单独的疾病。
Objective to study the mechanism of liver fibrogenesis and to find new non-invasive biomarkers.
目的研究肝纤维化的发生机制，寻找新的与肝纤维化相关的生物学标志物。
We know that hepatic stellate cells are still the major source of extracellular matrix during fibrogenesis.
我们知道肝星状细胞还是纤维化过程中细胞外基质的主要来源。
Objective to observe the differentiation of bone marrow stem cells in rat hepatic fibrogenesis environment into hepatocytes.
目的研究骨髓干细胞在肝纤维化形成环境中向肝细胞定向分化。
Objective To study the role of hepatic sinusoidal endothelium injury during hepatic fibrogenesis induced by dimethylnitrosamine (DMN) in rats.
目的研究肝窦内皮细胞损伤在二甲基亚硝胺大鼠肝纤维化形成中的作用。
Objective to study the role of the synthesis and degradation of collagen at the transcription level during liver fibrogenesis due to schistosomiasis japonica in rabbits.
目的从转录水平研究兔日本血吸虫病肝纤维化形成过程中胶原的合成与降解的规律。
Conclusions it was confirmed at transcription level that when the quantity of collagen synthesis was higher than that of collagen degradation liver fibrogenesis may be resulted in.
结论从转录水平证实胶原合成大于降解时可导致肝纤维化形成。
Fibrogenic cells, cytokines, growth factors, enzymes, and their inhibitors play a pivotal role in fibrogenesis. These will advance our recognition for pathogenic mechanisms of hepatic fibrosis.
在纤维形成过程中，促纤维形成细胞、细胞因子、生长因子、酶及其抑制因子发挥了十分重要的作用。
Fibrogenic cells, cytokines, growth factors, enzymes, and their inhibitors play a pivotal role in fibrogenesis. These will advance our recognition for pathogenic mechanisms of hepatic fibrosis.
在纤维形成过程中，促纤维形成细胞、细胞因子、生长因子、酶及其抑制因子发挥了十分重要的作用。
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