FGF-2 treatment resulted in lower aggrecan turnover compared to untreated controls under identical culture conditions.
与在同样培养条件下的对照组相比，FGF2处理导致更少的蛋白多醣的转化。
Monoclonal antibody is produced by immunizing animals with a recombinant protein corresponding to residues near the carboxy terminal region of human FGF receptor 2 protein.
该单克隆抗体是通过用重组蛋白免疫动物而制备的，该重组蛋白是人FGF受体2蛋白羧基末端区域的残基。
FGF-2 has been shown to retain the differentiation potential of monolayer expanded chondrocytic cells. However, its effect on nucleus pulposus cells is not known.
在单层培养的软骨细胞扩展中，FGF-2显示出可以保持分化的潜力，但此种作用在髓核细胞中尚不明确。
Conclusion Intra-arterial infusion of FGF-2 may significantly promote neovascularization and vascular perfusion in ischemic limbs, and lead to improvement of its blood supply.
结论经动脉内局部灌注f GF - 2可明显促进缺血肢体的血管生长和血液灌流，从而改善其局部血液供应。
Objective: to explore the temporal change on the expression of GDNF and FGF-2 in spared dorsal root ganglia after partial dorsal root rhizotomy and acupuncture.
目的：探讨部分背根切断及针刺后不同时相备用背根节gdnf、FGF - 2的表达变化。
FGF receptor 2 (D4H9) Rabbit mAb recognizes endogenous levels of total FGF receptor 2 protein. This antibody does not cross-react with other FGF receptor family members.
兔单抗能够识别内源性水平的总FGF受体2蛋白。该抗体不与其他FGF受体家族成员发生交叉反应。
FGF-2, a potent agonist of Egr-1, plays an important role vascular repair after injury.
作为早期生长反应基因-1的增效剂，纤维生长因子-2 在损伤后的血管修复中起着重要的作用。
FGF-2 action resulted in reduced actin stress fiber formation and migratory cell morphology, with no effect on cell proliferation.
FGF2可减少肌动蛋白张力丝的形成以及移行细胞的形态，但对细胞增殖没有作用。
Moreover, FGF-2 treatment resulted in increased sulfated proteoglycan synthesis and lower aggrecan turnover compared to untreated controls under identical culture conditions.
而且，与在同样培养条件下的对照组相比，FGF2处理导致硫酸化蛋白多醣的合成，以及更少的蛋白多醣的转化。
Moreover, FGF-2 treatment resulted in increased sulfated proteoglycan synthesis and lower aggrecan turnover compared to untreated controls under identical culture conditions.
而且，与在同样培养条件下的对照组相比，FGF2处理导致硫酸化蛋白多醣的合成，以及更少的蛋白多醣的转化。
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