Conclusions Fetal rat brain damage is made by LPS-induced intrauterine infection.
结论LPS致宫内感染可造成胎鼠脑损伤。
The distribution and content alternation of NPY in the fetal rat brain was studied immunohistochemically.
通过免疫组织化学方法观察胚胎大鼠脑组织神经肽Y的分布与含量变化。
Objective: To verify the effect of maternal folate deficiency (FD)on fetal rat brain development, and the change of DNA methylation.
目的：从分子水平探讨叶酸缺乏（FD）对胎鼠脑发育的影响。
Objective To investigate the effect of ant powder on the brain, liver and placenta of fetal rat with IUGR.
目的了解蚂蚁粉对宫内发育迟缓（IU GR）胎鼠的大脑，肝脏和胎盘的作用。
Finally, the biological activity of thes proteins to brain neuron cell of fetal rat were assayed by mean of cell biology method.
最后我们利用细胞生物学的方法，检测了这些蛋白对胎鼠大脑神经细胞的生物活性。
Method: Fetal rat microglia in brain was isolated and purified by lidocaine hydrochloride with shock culturing. The purification of microglia was detected by immunofluorescence and flow cytometry.
方法利用盐酸利多卡因注射液联合震荡培养法分离纯化胎大鼠脑内小胶质细胞，免疫荧光技术及流式细胞技术鉴定小胶质细胞的纯度。
Method: Fetal rat microglia in brain was isolated and purified by lidocaine hydrochloride with shock culturing. The purification of microglia was detected by immunofluorescence and flow cytometry.
方法利用盐酸利多卡因注射液联合震荡培养法分离纯化胎大鼠脑内小胶质细胞，免疫荧光技术及流式细胞技术鉴定小胶质细胞的纯度。
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