By the time of reperfusion, fetal brain damage increases progressively.
并随着再灌注时间的延长而脑损伤逐渐加重。
This heavy metal, which can also contaminate water and soil, can interfere with nearly every aspect of fetal development, causing brain and kidney damage, according to the CCHE.
铅这种重金属，也会污染水和土壤，而且，根据CCHE，铅几乎可以干扰到胎儿发育的各个方面，造成脑和肾的损伤。
Fetal brain cell damage that phenomenon, would lead to fetal body dysplasia.
这种胎儿脑细胞受损的现象，会导致胎儿全身发育不良。
Objective To investigate the neuroprotective effects of flunarizine (FNZ) and lamotrigine (LTG), and their combination on the hypoxic-ischemic brain damage in fetal rats.
目的研究氟桂利嗪(FNZ)、拉莫三嗪(ltg)及两药联合经孕鼠给药，对胎鼠宫内缺血缺氧性脑损害的保护作用。
Fetal inner ear by noise stimulation, the brain can damage parts of the region, and seriously affect the intellectual development.
胎儿内耳受到噪音的刺激，能使脑的部分区域受损，并严重影响智力的发育。
Conclusions Fetal rat brain damage is made by LPS-induced intrauterine infection.
结论LPS致宫内感染可造成胎鼠脑损伤。
Conclusion After severe intrauterine fetal distress, changes of free radicals occurred in brains firstly, which provides the proof of early treatment for neonatal hypoxia ischemia brain damage.
肝组织均未发生显著变化。结论宫内窘迫时，脑组织首先出现自由基变化，为新生儿缺血缺氧性脑损伤的早期防治提供了理论依据。
Conclusion After severe intrauterine fetal distress, changes of free radicals occurred in brains firstly, which provides the proof of early treatment for neonatal hypoxia ischemia brain damage.
肝组织均未发生显著变化。结论宫内窘迫时，脑组织首先出现自由基变化，为新生儿缺血缺氧性脑损伤的早期防治提供了理论依据。
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