Objective: To study the time-effect relationship of mice embryonic toxicity and teratogenicity induced by HCHO in vitro.
目的：探讨甲醛对植入后小鼠胚胎发育毒性及时间-效应关系。
Conclusion Disturbance of embryonic cell behavior and related gene expression played important roles in developmental toxicity caused by methylmercury.
结论胚胎细胞行为和相关基因表达异常在甲基汞的发育毒性作用机理中起重要作用。
Opuntia milpa alta flower has no maternal toxicity, embryonic and teratogenicity under the experimental conditions.
在本实验剂量和条件下仙人掌粉未见有母体毒性、胚胎毒性和致畸作用。
Objective to explore the mechanism of embryonic developmental toxicity of methyl mercury at cellular and gene levels.
目的从细胞和基因水平揭示甲基汞的发育毒性作用机理。
Conclusions Fluoride could result in embryonic growth retardation and potential teratogenic toxicity. These effects might be due in part to decrease in GSH activity and membrane lipid fluidity.
结论氟有潜在的致畸性和胚胎毒性，胚胎组织gsh活性和卵黄囊细胞膜脂流动性降低可能在氟致胚胎发育毒性中起重要作用。
Conclusions Fluoride could result in embryonic growth retardation and potential teratogenic toxicity. These effects might be due in part to decrease in GSH activity and membrane lipid fluidity.
结论氟有潜在的致畸性和胚胎毒性，胚胎组织gsh活性和卵黄囊细胞膜脂流动性降低可能在氟致胚胎发育毒性中起重要作用。
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