Conclusion The AP-1 DNA binding ability was markedly increased in liver in burned mice.
结论烧伤早期引起小鼠肝组织AP - 1的DNA结合活性显著增强。
While heat shock promoted the phosphorylation and nuclear translocation of HSF1, the ability of this nucleus-localized HSF1 to form the DNA-binding trimer was reduced.
HSF1的亚细胞定位分析显示，HSF1主要存在于年轻细胞胞质中，热刺激促使三体形成和核转移；
While heat shock promoted the phosphorylation and nuclear translocation of HSF1, the ability of this nucleus-localized HSF1 to form the DNA-binding trimer was reduced.
HSF1的亚细胞定位分析显示，HSF1主要存在于年轻细胞胞质中，热刺激促使三体形成和核转移；
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