• We apply complex network theory to analyze the topological properties of DLBCL gene network.

    应用复杂网络理论研究DLBCL基因网络的拓扑结构特性。

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  • We studied a series of 16 patients with nodal DLBCL occurring in patients between 10 and 18 years of age.

    在此,我们研究了一组少年儿童的结节状DLBCL,患者共16例,年龄介于10至18岁。

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  • This review summarizes the morphological, immunophenotypic, cytogenetic and molecular genetic features of DLBCL relevant to the precise prognostic previsions.

    作者综述DLBCL的形态学、免疫组化、分子生物学特点及与预后的关系。

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  • As only 40% of patients benefit from chemotherapy, researchers suspected that DLBCL and other lymphomas could be caused by a variety of cell types gone haywire.

    只有40%的病人得益于化疗,研究人员怀疑DLBCL和其它淋巴瘤可能是不同的类型的细胞出问题而形成的。

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  • The project of alternate half-body irradiation combined with R-CHOP to treat DLBCL has effect on apparente synergia and attenuation and could improve the patient's quality of life.

    交替半身照射联合美罗华治疗弥漫性大B细胞淋巴瘤有明显的增效、减毒及改善患者生活质量的作用。

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  • PURPOSE: the current standard therapy for patients with diffuse large B-cell lymphoma (DLBCL) is rituximab plus cyclophosphamide, doxorubicin, vincristine, and prednisone (R-CHOP).

    背景:目前,弥漫大b细胞性淋巴瘤(DLBCL)的标准治疗方案为利妥昔单抗联合CHOP方案化疗(R - CHOP)。

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  • These results demonstrate that CARD11 is a bona fide oncogene in DLBCL, providing a genetic rationale for the development of pharmacological inhibitors of the CARD11 pathway for DLBCL therapy.

    这些结果表明在DLBCL中,CARD11是真正的癌基因,对DLBCL治疗中用药物抑制CARD11通路提供了遗传学理论。

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  • These results demonstrate that CARD11 is a bona fide oncogene in DLBCL, providing a genetic rationale for the development of pharmacological inhibitors of the CARD11 pathway for DLBCL therapy.

    这些结果表明在DLBCL中,CARD11是真正的癌基因,对DLBCL治疗中用药物抑制CARD11通路提供了遗传学理论。

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