CONCLUSION Adenosine could protect the damage of ischemia reperfusion.
腺苷对大鼠肾脏缺血再灌注损伤有保护作用。
OBJECTIVE To investigate the protective mechanism of adenosine in renal damage of ischemia reperfusion.
探讨腺苷对肾脏缺血再灌注损伤的保护作用机制。
Objective:To study the preventive effect of damage of ischemia reperfusion in rat liver by applying Shenmai Injection.
目的：研究参麦注射液对肝缺血再灌注损伤的保护作用。
These results showed that the energy metabolism dysfunction was the initial stage, and the damage of oxygen free radicals was the major factor in ischemia-reperfusion injury.
这些结果说明线粒体能量代谢障碍是缺血再灌注损伤的始动环节，而氧自由基是造成损伤的主要因素。
The said kidney disease in this invention includes of chronic kidney disease, diabetic nephropathy, kidney damage coursed by acute ischemia reperfusion.
本发明所述的肾脏疾病包括慢性肾脏疾病、糖尿病肾病、急性缺血再灌注导致的肾脏损伤。
Conclusion In the process of liver transplantation, the ischemia reperfusion damage may lead to hepatic microcirculation disturbance, which is a major cause of graft failure.
结论肝移植过程中，缺血再灌注损伤可造成肝脏微循环紊乱，是供肝失活的一个重要原因。
Objective to investigate the relationship between the apoptosis of intestinal epithelium and characteristics of intestinal mucosal ischemia-reperfusion damage and repair after injury in rats.
目的探讨小肠黏膜缺血损伤及再生修复的特征及规律，以及与肠上皮细胞凋亡的关系。
Aim to investigate the protective effects of MLT (melatonin) on acute ischemia-reperfusion induced myocardium damage in rats in vivo.
目的探讨外源性褪黑素(MLT)对在体大鼠心肌急性缺血再灌注损伤的保护作用。
Aim: Damage of endothelial cells and adhesion of leukocytes and platelets were studied in rat mesentery microvessels after ischemia reperfusion.
目的：在活体上探讨缺血再灌后血管内皮细胞损伤及白细胞、血小板与内皮之间粘附的变化。
These results suggest that oxygen free radicals may damage the activity of cytochrome oxidase, the reduction of which may play an important role in the injury of renal ischemia and reperfusion.
提示氧自由基可能损害细胞色素氧化酶活性，细胞色素氧化酶活性降低在肾缺血再灌流损伤中可能起重要作用。
Objective To explore the survival mechanism of hippocampal neurons after the damage of hypoxic ischemia and reperfusion of the brain.
目的探讨缺氧缺血再灌注后海马神经元的存活机制。
Objective The effects of YVAD-FMK on neuron damage after complete cerebral ischemia-reperfusion injury in rats were studied.
目的研究YVAD - FMK对大鼠全脑缺血再灌注后神经元的影响。
Conclusion Propofol may inhibit hypoxia in the brain and the apoptosis of nerve cells in result of protecting the cerebral ischemia and reperfusion damage in rats.
结论异丙酚可能通过抑制大脑缺氧，抑制神经元的凋亡，从而对大鼠局灶性脑缺血-再灌注损伤发挥保护作用。
Conclusion: Damage of epiphysis could be aggravated after ischemia and reperfusion injury.
结论：缺血再灌注可加重幼年兔股骨头骨骺骨细胞损伤。
Result: PNS could reduce infarct volume of cerebral ischemia-reperfusion injury in rats, reduce the extent of the damage in blood-brain barrier significantly.
结果：三七总皂苷可有效降低脑缺血再灌注损伤大鼠的脑梗死体积，明显减轻脑缺血区血脑屏障破坏的程度。
Result: PNS could reduce infarct volume of cerebral ischemia-reperfusion injury in rats, reduce the extent of the damage in blood-brain barrier significantly.
结果：三七总皂苷可有效降低脑缺血再灌注损伤大鼠的脑梗死体积，明显减轻脑缺血区血脑屏障破坏的程度。
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