METHODS:The models of cerebral ischemia reperfusion injury and senile dementia mice caused by D galactose were used.
方法：采用脑缺血再灌注损伤及D 半乳糖所致痴呆小鼠模型。
Objective to observe the changes of morphology and biochemical parameters in rats with D-galactose-induced skin ageing.
目的观察d -半乳糖诱导皮肤衰老大鼠的皮肤形态学改变及与衰老相关的生化指标变化。
Conclusion It was suggested that Pyracanthy had some antioxidation and might prolong the aging process of mice model induced by overdose of D-galactose.
结论火棘有一定的抗氧化作用，并能延缓由D -半乳糖所致衰老小鼠的衰老过程。
Calculate the percentage composition of the two forms of D-galactose at equilibrium.
计算达到平衡时这两种形式的D -半乳糖的百分含量。
AIM: to observe the protective effect of extract from maize plumule against lipid peroxidation in drosophila induced by D-galactose.
目的：观察玉米幼芽提取物对d -半乳糖引起果蝇脂质过氧化的拮抗作用。
Results Compared with the D-galactose induced mouse model, apigenin could improve the ability of escaping electrical stimulation significantly(P<0.01).
结果腹腔注射芹黄素的小鼠学习能力和记忆保持率均显著优于模型对照组（P<0.01）。
Objective to investigate the effects of Pingmaitong granules on free radical metabolism of D-galactose-induced subacute aging model.
目的探讨平脉通颗粒对D -半乳糖所致亚急性衰老模型自由基代谢的影响。
Methods: Senile rats model was established by adopting the method that injected D-galactose subcutaneously in nape.
方法：采用D -半乳糖颈后皮下注射的方法，造成亚急性衰老动物模型。
Objective: to study the effect of Dunhuang Weicifang delaying senility to D-galactose presenile model mice.
目的：研究敦煌韦慈方对d半乳糖所致衰老模型小鼠的延缓衰老作用。
Methods Subacute senile mouse model was established by injection of D-galactose.
方法通过注射d半乳糖建立亚急性衰老小鼠模型。
Objective To observe the effect of carboxymethyl chitosan (CMC) on the lifespan of Drosophila melanogaster and D-galactose-induced aging in mice.
目的观察羧甲基壳聚糖（CMC）对黑腹果蝇寿命的影响及其对D-半乳糖致亚急性衰老小鼠的抗衰老作用。
Methods Subacute senile mice model was established by injection of D-galactose.
方法通过注射D半乳糖建立亚急性衰老小鼠模型。
Objective To study the effect of oxygen stress induced by D-galactose unite with hyperlipoidemia on hemorheology and carotid blood perfusion in rats.
目的观察D-半乳糖诱发氧应激及实验性高脂血症对大鼠血液流变特性的影响。
Objective to study the effects of chickpea protein hydrolysate (CPH) in different doses on of antioxidant abilities in serum, liver and heart in aged mice induced by D-galactose.
目的研究不同剂量鹰嘴豆蛋白酶解物(CPH)对d -半乳糖致衰小鼠血清、心脏和肝脏抗氧化能力的影响。
Aim By using natural aging mice and D-galactose-induced subacute aging mice, Anti-aging effects of GAP were studied and the mechanism of the effects was discussed.
目的研究白果清蛋白(GAP)对自然衰老小鼠及半乳糖致亚急性衰老模型小鼠的延缓衰老作用，并探讨作用机制。
To study the antioxidant effect of the hawthorn extract on D-galactose induced aging mice model.
探讨山楂提取物对d -半乳糖致衰小鼠抗氧化系统的影响。
D-galactose was used to build aging model.
利用D-半乳糖建立衰老模型。
Objective to observe the effects of zinc-deficiency (ZD) on antioxidation system and repair of DNA damage and of hepatic cells by establishing an aging mice model induced with D-galactose.
目的通过d半乳糖诱导小鼠衰老模型，探讨缺锌对衰老小鼠抗氧化系统和肝脏dna损伤与修复的影响。
Conclusions the tetramethylpyrazine can significantly improve central cholinergic system function, and thus enhance the learning and memory ability in D-galactose-lesioned mice.
结论川芎嗪可明显提高模型小鼠的学习记忆能力，改善中枢胆碱能系统功能。
AIM: to investigate the inductive effects of D-galactose, which can induce cell aging of fibroblasts and neurons, on the modeling senescence of cardiac myocytes in rats.
目的：观察可诱导成纤维细胞、神经元细胞衰老的D -半乳糖对心肌细胞拟衰老的诱导作用。
Objective to study the effect of Renshen Yangrong decoction (RYD) in protecting morphology and structure of mitochondria in brain and liver of D-galactose induced subacute senile mice.
目的研究人参养荣汤对亚急性老化小鼠肝、脑细胞线粒体形态结构变化的保护作用。
Objective: to observe the protective effect of tremella polysaccharides (TP) on mouse aging model induced by D-galactose.
目的：观察银耳多糖对D -半乳糖所致衰老小鼠的作用。
Conclusion: Erzhi Pill can effectively resist the skin senility of the mouse with subacute senility resulting from D-galactose.
结论：二至丸能有效防止D -半乳糖致衰老小鼠的皮肤衰老。
The loose arrangement of collagen and elastic fibers was evident in D-galactose group. These features of the rats in D-galactose group were similar to those in the ageing skin of human beings.
结果D -半乳糖组大鼠表皮变薄，胶原纤维及弹力纤维减少，排列疏松，呈现与人类皮肤衰老相似的表现。
SD rats were randomly assigned to five groups as follows: saline control group, D-galactose group, DHEA-treated normal group, vehicle control group and DHEA-treated senescent group.
衰老模型实验：SD大鼠随机分为对照组、D-半乳糖组、正常用药组、溶媒对照组和药物治疗组。
Objective: To observe the actions and mechanism of Erzhi Pill for resisting the skin tissue senility of the mice with subacute senility resulting from D-galactose.
目的：观察二至丸对D-半乳糖致亚急性衰老小鼠抗皮肤衰老的作用及机制。
Conclusion: Puerariae can significantly inhibit glycation reaction in rats induced by D-galactose.
结论：葛根素对d -半乳糖诱导的大鼠糖基化反应具有明显的抑制作用。
Methods: Y-electronic maze was used to study the behavior of dementia rats induced by D-galactose and QA.
方法：采用D-半乳糖、喹啉酸致痴呆大鼠模型，使用大鼠Y型电迷宫法，研究药物对大鼠学习记忆的影响；
Methods: Y-electronic maze was used to study the behavior of dementia rats induced by D-galactose and QA.
方法：采用D-半乳糖、喹啉酸致痴呆大鼠模型，使用大鼠Y型电迷宫法，研究药物对大鼠学习记忆的影响；
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