• CONCLUSION: GLM could enhance the cytotoxic effect of ADM on drug resistant cell and increase intracellular drug concentration in a concentration dependent fashion.

    结论GLM提取物提高ADM细胞细胞毒作用增加了耐药细胞药物浓度,且呈剂量依赖性。

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  • The results showed at the same test concentration, the extent of cytopathic effect (CPE) among different cell lines was different, the sensitivity difference was obvious.

    结果显示同一浓度处理下,不同细胞系细胞病变程度不同,敏感性差异很大。

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  • Results: The main influential factors are the concentration of testee and its time of effect on the cell, the cell cycle in SCGE.

    结果:受试物浓度及其细胞作用时间、细胞周期等是SCGE实验中的主要影响因素

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  • Objective:To obsreve the effect of cell concentration on hemopoietic stem cell cryopreservation.

    目的观察细胞浓度造血干细胞冻存效果影响。

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  • The lobeline up-regulates the concentration of synapse intermission DA by inhibiting the transport of DAT, so the apoptosis speed of a nerve cell can be stopped or retarded with little adverse effect.

    贝林可通过抑制多巴转运蛋 白转运功能而上调突触间歇多巴胺浓度能够阻止减缓神经细胞凋亡速度,而且副 作用

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  • Concentration of LMW NTS had effect on mitochondrial succinodehydrogenase activity of tumor cell, lower concentration showed a promotion action, higher concentration showed a slight inhibition action.

    不同浓度LMW NTS肿瘤细胞线粒体琥珀酸脱氢酶活力的影响不同,浓度时表现促进作用而高浓度时表现为轻度抑制作用。

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  • The effect of thymic peptide hormone on the changes of PNA receptors on the cell surface of mouse thymocyte was found to be both concentration and time dependent.

    我们研究了胸腺激肽与胸腺细胞温育时间、不同剂量及酸、碱、热和酶处理后的胸腺激肽PNA受体影响

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  • Methods it was observed that different concentration anti_digoxin antiserum effect on endogenous digitalis_like factor and cell membrane ATPase activity in hypoxic myocardium model.

    方法制备心肌组织缺氧损伤模型观察不同剂量的地高辛抗血清对缺氧损伤心肌组织内源性洋地黄样因子水平心肌细胞膜atp酶活性影响

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  • METHODS: It was observed that different concentration of anti-digoxin antiserum effect on endoxin and cell membrane ATPase activity in hypoxic myocardium model.

    方法: 制备心肌组织缺氧模型观察不同剂量地高辛抗血清缺氧损伤心肌组织内洋地黄素水平心肌细胞膜ATP酶活性影响

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  • METHODS: It was observed that different concentration of anti-digoxin antiserum effect on endoxin and cell membrane ATPase activity in hypoxic myocardium model.

    方法: 制备心肌组织缺氧模型观察不同剂量地高辛抗血清缺氧损伤心肌组织内洋地黄素水平心肌细胞膜ATP酶活性影响

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