Abjective: To establish the rabbit arteriosclerosis mold caused by chronic intermittent hypoxia, and probe into the correlative malgenic mechanism.
目的：建立慢性间歇缺氧致动脉粥样硬化兔模型，探讨相关致病机制。
Objective:To establish the rabbit arteriosclerosis mold caused by high-adiposed diet under the condition of chronic intermittent hypoxia, and probe into the correlative mechanism.
目的：在慢性间歇缺氧的条件下，用高脂饮食方法建立兔的动脉粥样硬化模型，探讨相关致病机制。
The results suggest that KV inhibition was significantly attenuated by chronic intermittent hypoxia, and this effect may be a critical mechanism of the body hypoxic acclimatization.
上述观察结果提示慢性间歇性低氧减弱急性缺氧对KV的抑制，这可能是机体低氧适应的一种重要机制。
Conclusion Chronic intermittent hypoxia can increase rat liver microsomal ERD and ANH activities. The mechanism is related to up-regulation of CYP3A2 and CYP2E1 expression at the transcriptive levels.
结论慢性间断性低氧能明显增加大鼠肝脏erd (CYP3A2)和ANH (CYP2E1)活性，其机制可能与其在转录水平上提高肝脏cyp3a2和CYP2E1的基因表达水平有关。
This article reviews previous and recent studies that have examined the changes in the expression of proteins in response to chronic sustained and intermittent hypoxia.
本文综述先前及近期关于慢性持续低氧及间断缺氧情况下蛋白质表达改变的研究。
The effect, of chronic intermittent hypobaric hypoxia on pulmonary vascular reactivity to acute hypoxia in. rats and its relationship to histamine have been studied.
作者研究了慢性间断低压缺氧对大鼠肺血管反应性的影响及其与组胺的关系。动物经慢性缺氧后发生慢性肺动脉高压，其肺血管对急性缺氧的反应性降低。
The effect, of chronic intermittent hypobaric hypoxia on pulmonary vascular reactivity to acute hypoxia in. rats and its relationship to histamine have been studied.
作者研究了慢性间断低压缺氧对大鼠肺血管反应性的影响及其与组胺的关系。动物经慢性缺氧后发生慢性肺动脉高压，其肺血管对急性缺氧的反应性降低。
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