This research analysed the function of CEO2, a potential transcription regulator factor, and the molecular mechanism of heavy metal injury.
本研究对拟南芥一个可能的转录调节因子ceo2功能及其介导重金属伤害的分子机制进行了分析。
The superfine powders of CeO2 were modified using cheap stearic acid and dispersion-liquid made by our laboratory, to improve their dispersibility in polypropylene (PP).
为提高纳米氧化铈粉体在高聚物中的分散性能，以廉价的硬脂酸和实验室自制的分散液，对超微氧化铈粉体进行表面改性。
By histochemical detection of GUS activity, high levels of GUS expression were found in metabolism tissues such as seedling, stigma and anther, and CEO2 maybe localized in nuclear.
GUS组织化学分析发现CEO2主要在代谢比较旺盛的部位如幼苗、柱头及其花药等组织表达，CEO2可能定位于细胞核。
Adding the different nanometer CeO2 content to alkali iron powder electrode, the diffusible hydrogen content, low-temperature impact and chemical composition of the weld were tested.
在碱性铁粉焊条药皮中添加不同量的纳米稀土氧化物粉体，对焊缝进行扩散氢含量、低温冲击及化学成分的测试和分析。
Adding the different nanometer CeO2 content to alkali iron powder electrode, the diffusible hydrogen content, low-temperature impact and chemical composition of the weld were tested.
在碱性铁粉焊条药皮中添加不同量的纳米稀土氧化物粉体，对焊缝进行扩散氢含量、低温冲击及化学成分的测试和分析。
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