• Understanding cellular response to environmental stress has broad implications for human disease.

    细胞对环境应激的反应与人类疾病有广泛的联系。

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  • This review briefly summarizes the current progress in cellular response to DSB.

    本文将简单概述当前对DSB细胞应答系统的研究进展。

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  • Similarly, the cellular response to the environmental mutagens is not always initiated by DNA damage.

    同样地,环境诱变剂诱发的细胞应答反应也不只是单纯地由DNA损伤所触发的。

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  • Now there are two ways in endotoxin induced cellular response: the indirect way of cytokines and the direct way.

    目前认为内毒素介导细胞的反应通过两个途径:细胞因子的间接作用及对细胞的直接作用。

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  • Novel IFN receptor 1 binding proteins, DNA encoding them, and method of modulating cellular response to interferons.

    新型IFN受体1结合蛋白质,编码它们的DNA,以及调节细胞对干扰素的反应的方法。

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  • Novel IFN receptor 1 binding proteins, DNA encoding them, and methods of modulating cellular response to interferons.

    新型IFN受体1结合蛋白质,编码它们的DNA,以及调节细胞对干扰素的反应的方法。

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  • The p53 tumor suppressor protein plays a major role in cellular response to DNA damage and other genomic aberrations.

    p53肿瘤抑制蛋白在细胞响应DNA损伤和其它基因组异常的过程中发挥重要作用。

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  • In particular, the researchers found that two key genes were affected in the cellular response to decreased glucose consumption.

    特别是,研究人员发现,葡萄糖消耗下降会影响细胞的两个关键基因。

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  • Objective to explore the cellular response of human senescent diploid fibroblasts (2bs) to the DNA damage caused by methyl methanesulfonate (MMS).

    目的观察衰老的人胚肺二倍体成纤维细胞(2bs)对烷化剂甲磺酸甲酯(MMS)诱导的DNA损伤的应答。

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  • An important part of the cellular response to DNA damage is checkpoint activation - checkpoint kinases CHK1 and CHK2 phosphorylate key proteins to elicit cell-cycle blocks.

    细胞对DNA损伤作出应答的一个重要过程是检查点活化(检查点激酶CHK1和CHK2使关键蛋白磷酸化,从而阻止正常的细胞周期。)

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  • The contents mainly include mechanical stimulation signal, cells feeling the mechanical stimulation signal, force transduction mechanisms and cellular response to mechanical stimulation.

    研究内容主要有力学刺激信号、细胞对力学刺激信号的感受及转导机制和细胞受力后的反应三部分内容。

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  • Therefore, the aim of this study was to focus on the cellular response and events of dermal contraction and epidermal re-epithelialization during wound healing under the influence of AgNPs;

    因此,本研究的目的是纳米银粒子存在情况下创面愈合时真皮收缩和表皮再生过程中细胞的应答和变化。

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  • Therefore, the aim of this study was to focus on the cellular response and events of dermal contraction and epidermal re-epithelialization during wound healing under the influence of AgNPs;

    因此,本研究的目的是纳米银粒子存在情况下创面愈合时真皮收缩和表皮再生过程中细胞的应答和变化。

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