The roles of protein tyrosine phosphatase SHP 2 in breast cancer (MCF 7) cell adhesion and migration were investigated.
探讨蛋白酪氨酸磷酸酶SHP2在乳腺癌细胞MCF7的移动及粘附中的作用。
The cell form was observed by light invert microscope. The adhesive and invasive ability was detected by cell adhesion and migration experiments.
以细胞迁移实验和粘附实验观察细胞侵袭和粘附能力的影响。
Ha on the surface of macrophages and ha substratum increased cell adhesion and migration, addition of free ha into the medium decreased cell adhesion.
巨噬细胞表面ha和底物HA增加细胞黏附，促进细胞迁移，但游离HA降低细胞黏附。
As one of the members of heparan sulfate proteoglycans, Glypican-3 (GPC3) participates in the process of cell proliferation, differentiation, adhesion and migration.
磷脂酰肌醇蛋白聚糖3是硫酸乙酰肝素糖蛋白家族中的一员，它参与调控细胞增殖、分化、黏附和迁移等过程。
Scaffold protein IQGAP1 has been found to bear close relationship with many important cell activities, such as adhesion and migration.
支架蛋白iqgap1发现能与许多重要的细胞活动紧密相关，如黏附和迁移。
Then cell morphological changes are induced, such as polarity, protrusion, and adhesion, which lead to the migration of endothelial cell at last.
这些级联反应引起细胞形态学上的进一步变化，如极化、突起、黏附，最终导致细胞的迁移运动。
Many adhesive proteins contain the Arg Gly Asp (RGD) sequence which plays an important role in cell adhesion, migration and growth.
RGD是许多粘附蛋白所共有的高度保守氨基酸序列，这一序列在介导细胞粘附、迁移及生长方面起重要作用。
At the same time, PTEN has the high homology with tensin and auxilin, PTEN may suppress cell migration, spreading, and adhesion formation.
同时PTEN氨基酸序列与张力蛋白和辅助蛋白高度同源，对细胞迁移、扩展、局灶黏附活动有抑制作用。
Objective To study effects of the exogenous wild-type PTEN tumor suppressor gene on in vitro adhesion, migration and invasion of the highly metastatic mucoepidermoid carcinoma cell line M3SP2.
目的探讨外源野生型PTEN抑癌基因对人高转移性黏液表皮样癌细胞系m 3sp2体外黏附、迁移和侵袭特性的影响。
Conclusion the exogenous wild-type PTEN gene has significant effects of anti-adhesion, anti-migration, and anti-invasion on the highly metastatic mucoepidermoid carcinoma cell line M3SP2.
结论外源性pten抑癌基因对高转移性黏液表皮样癌细胞系m 3sp2体外黏附、迁移和侵袭具有抑制作用。
CONCLUSIONS RD can result in increased expression of several adhesion molecules known to be associated with cell proliferation and migration, and this change can be reversed by retinal reattachment.
结论：视网膜脱离会导致RPE细胞上几种重要的粘附分子的表达增加，视网膜复位可以逆转这种变化。
CONCLUSIONS RD can result in increased expression of several adhesion molecules known to be associated with cell proliferation and migration, and this change can be reversed by retinal reattachment.
结论：视网膜脱离会导致RPE细胞上几种重要的粘附分子的表达增加，视网膜复位可以逆转这种变化。
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