Objective To explore the protective effect of Yam polysaccharide on CCL4-induced liver injury.
目的：探讨山药多糖对小鼠ccl4肝损伤的保护作用及可能作用机制。
Methods CCL4 was injected intraperitoneally or subcutaneously to make rat liver fibrosis model.
方法分别利用CCL_4腹腔注射和皮下注射的方法制备大鼠肝纤维化模型。
The fluorescence quenching of aromatic oxazole derivatives by CCl4 in different solvents was studied.
本工作对唑类化合物在不同溶剂中的荧光被四氯化碳的猝灭问题进行了研究。
METHODS: Cirrhosis model was induced in male SD rats by injection of 60%ccl4 oily solution subcutaneously.
方法：用60%四氯化碳油性溶液皮下注射SD大鼠制造肝硬化大鼠模型。
This study was designed to evaluate the effects of tea seed oil on CCl4-induced acute hepatotoxicity in rats.
本研究旨在探讨影响茶叶籽油对四氯化碳诱导的急性肝损伤的影响。
The protective effect of Xiongdan tea on mice with chronic hepatic injury induced by CCl4 and D-Galn was studied.
研究了熊胆茶的肝保护作用及其抗炎免疫作用。
METHODS The acute liver injury models were induced by hypodermic injection of carbon tetrachloride( CCl4) in mice.
目的研究肝舒胶囊对大、小鼠急性肝损伤的保护作用。
Objective: to study the protective effect in CCl4-induced liver injury by organoselenium from Se-enriched lactobacillus.
目的：研究乳酸菌源有机硒保护肝损伤组织脂质过氧化反应及其机制。
Methods: on animal with liver injured by Ccl4 to make protecting liver and dropping ALT test and immunity function test.
方法：对实验性动物肝损伤进行保肝降酶和免疫功能研究。
Objective: To study the protective effects of powder of compound XuanJun capsules against the chemical liver injury in mice induced by CCL4.
目的：研究复方玄驹胶囊对小鼠化学性肝损伤的保护作用。
Now in China GB/T16488-1996 regards infrared spectrometry as the first method to determine oil centent. CCL4 is the extractant in this method.
目前国标GB/T16488—1996测油以红外分光光度法为第一方法，该法用CCL4 作萃取剂。
Results In CCL4-treated mice, the liver surface was micronodular, and the liver showed the histologically characteristic features of cirrhosis.
结果小鼠经ccl。注射后肝脏表面出现细小结节，镜-下呈现肝硬化的组织学特征。
Conclusion The cirrhotic model in rats were induced by using alcohol interval intragastrically before CCL4 injection showed a high survival rate.
结论在进行CCL4皮下注射前用乙醇作间歇性灌胃可以明显提高大鼠肝硬变模型的存活率。
This experiment objected to explore the roll and significance of GLN in CCL4 induced acute hepatic injury, to provide some experiment basements for the clinical use of GLN.
本实验通过四氯化碳（CCL4）致大鼠急性肝损伤模型，研究GLN在急性肝损伤中尤其是肝细胞凋亡和胀亡的保护作用及意义，为GLN的临床应用提供实验依据。
The tests of changes in resistivity under different densities of acetic acid and CCl4 liquor have been made in the laboratory, and the law of abnormal distribution is pointed.
在实验室进行了醋酸溶液和四氯化碳溶液不同浓度下的电阻率变化测试，指出了异常分布的规律。
The quantity of CCL4 is decreased to the lowest limit in the paper by improving pretreatment, and a simpler way to wash apparatus is also provided to reach the request of assay.
本文以最大限度地减少四氯化碳的用量和操作步骤出发，提出一套操作性强的样品预处理方法。
In the normal tissues of grass carp, CCL4 gene had the highest expressional level in spleen, followed by heart, muscle, gill, liver, and little amount in intestine and kidney, trace amount in brain.
正常组织中，CCL4基因在草鱼脾脏表达量最高，心、肌、鳃、肝中次之，肠和肾中有少量表达，在脑中有痕量表达。
In the normal tissues of grass carp, CCL4 gene had the highest expressional level in spleen, followed by heart, muscle, gill, liver, and little amount in intestine and kidney, trace amount in brain.
正常组织中，CCL4基因在草鱼脾脏表达量最高，心、肌、鳃、肝中次之，肠和肾中有少量表达，在脑中有痕量表达。
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