The process of angiogenesis is a cascade reaction.
HCC诱导的血管新生过程是一系列的级联反应。
The development and trial of neuroprotective drugs have almost been launched in all links of ischemic cascade reaction.
几乎对缺血级联反应的所有环节都已进行过神经保护药物的开发和试验。
So how to inhibit complement from activating and interrupt amplification of cascade reaction may be direction of treatment of AP.
因此如何抑制补体激活，阻断其扩大的级联反应成为治疗急性胰腺炎的突破口。
In addition, a great deal of inflammatory factors can activate the expression of ICAM-1 protein and lead to inflammatory cascade reaction.
另外，许多炎性因子可激活ICAM - 1蛋白表达，导致炎症的级联反应。
BACKGROUND: Maintenance and activation of cascade reaction influence t cell proliferation or transformation into nonreactive state even apoptosis.
背景：启动维持并调节活化级联反应，决定了T细胞是活化增殖或转变为无反应状态甚至凋亡。
To study rudimentarily the role of IL-18 in acute immunity hepatic injury, and the important of blocking cascade reaction of cytokines by exerting IL-18 antibody in early stage.
通过动物实验，初步探讨IL-18在急性免疫性肝损伤中的意义，及早期使用IL-18抗体及时阻断细胞因子级联反应对保护肝组织的重要性。
Conclusion Mild hypothermia has a protective role in cerebral ischemic tissue and inhibits the inflammatory cascade reaction, which may be one of the neuroprotective role mechanisms.
结论亚低温对缺血脑组织具有保护作用，对炎症级联反应的抑制可能是其发挥脑保护作用的重要机制之一。
The mechanisms contributing to the brain damage have not yet been well clarified. The mechanisms may be related with ischemic cascade reaction, ischemic reperfusion and cell apoptosis.
自由基在继发性损伤中的具体机制尚未完全清楚，可能与缺血级联反应、缺血再灌注和细胞凋亡有关。
Objective: to assess emodin antagonism to cerebral ischemia injury, and to discuss the mechanism of emodin inhibiting the inflammatory cascade reaction from the levels and expressions of cytokines.
目的：评价大黄素抗脑缺血损伤作用，并从细胞因子水平及表达方面探讨其抑制炎性级联反应机制。
Conclusion Mannitol has a protective effect on rabbits with ALI through effecting various cascade reaction of inflammatory injury and down regulating of the generation and expression of CEC and IL-8.
结论甘露醇可能通过影响炎症损伤级联反应的多个环节，抑制CEC、IL - 8等多种效应因子的产生和表达，从而对ALI发挥防治作用。
The nuclear reaction is calculated using intra-nuclear cascade model.
核反应过程采用核内级联模型。
Activation of leukocytes after PTCA can trigger a serial cascade of pathophysiologic reaction, releasing some inflammation intermediates lead to restenosis.
PTCA后白细胞的激活可能触发一系列病理生理联级反应，释放某些炎症介质，促进血管平滑肌增殖导致再狭窄；
Activation of leukocytes after PTCA can trigger a serial cascade of pathophysiologic reaction, releasing some inflammation intermediates lead to restenosis.
PTCA后白细胞的激活可能触发一系列病理生理联级反应，释放某些炎症介质，促进血管平滑肌增殖导致再狭窄；
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