In this text, we reviewed the effect of atorvastatin on the treatment of cardiac myocyte hypertrophy.
本文从心肌细胞肥大这个角度阐述阿托伐他汀对心室重塑的影响。
Results of ultrastructure of cardiac myocyte: Apoptosis could be observed in myocardial cell in diabetic group.
心肌细胞超微结构观察结果：糖尿病组心肌组织内可见凋亡的心肌细胞。
METHODS: Patch clamp technique (whole cell recording) was used to record L-Ca2 + current in single cardiac myocyte.
方法：用全细胞膜片箝记录心室肌细胞的L -钙电流。
Objective: to investigate the role of endoplasmic reticulum stress in induction of cardiac myocyte apoptosis in viral myocarditis.
目的：探讨内质网应激反应对于诱导病毒性心肌炎心肌细胞凋亡的作用。
Conclusion: These findings suggest endoplasmic reticulum stress may contribute to the cardiac myocyte apoptosis in viral myocarditis.
结论：内质网应激反应可能对于诱导病毒性心肌炎的心肌细胞凋亡有一定作用。
Objective to investigate the effect of HOE642 on cardiac myocyte apoptosis of the heterotopic heart transplantation of rat non heart-beating donors.
目的研究HOE642对热缺血死亡大鼠颈部异位停跳心脏供体心肌细胞凋亡的作用。
In traditional thoughts, apoptosis doesn't exist in the mature cells which doesn't have regenerative capacity, such as cardiac myocyte and nerve cells.
传统的观点认为细胞凋亡不发生于诸如成熟的心肌细胞、神经细胞等不具备再生能力的细胞中。
Results: Research indicated that Leptin and Leptin receptor (OB-Rb) was closely related to the pathogenesis of Hypertensive Cardiac Myocyte Hypertrophy.
结果：研究表明瘦素和瘦素受体与高血压心肌肥厚的发病机制有密切关系。
In the article the effect of thyroid hormones was reviewed on cardiac ion channels and transports, so as to explain the changes of the action current on cardiac myocyte.
本文综述了甲状腺素对心脏离子通道和转运装置的作用，解释了其引起心肌细胞动作电位变化原因。
Study of mechanism of medicine actions, by quantitative analysis of cultured cardiac myocyte, is one of the cutting edge researches in myocyte dynamics and molecular biology.
通过对离体培养的心肌细胞特征参数的定量分析，从细胞力学和分子生物学的角度研究药物的作用机理，是目前生物医学工程学科的前沿性课题。
The major goal to improve post-infarction prognosis would be the stimulation of neovascularization and the enhancement of regeneration of cardiac myocyte within the infarct area.
因此如果能够采取治疗措施促进梗死区灌注血管重建和心肌细胞再生，将会明显改善心梗患者的预后。
Meathod: The experiment adopted density gradient centrifuging to separate PBMC of 44 patients with CHF and 20 normal person and enzyme digestion to separate neonatal rat cardiac myocyte.
方法：密度梯度离心法分离44例CHF患者和20例正常人PBMC，酶消化和差速贴壁法分离乳鼠心肌细胞。
Digoxin directly inhibits the sodium-potassium ATPase pump in the membrane of the cardiac myocyte, causing an increase in intracellular sodium and calcium which increases myocardial contractility.
地高辛直接抑制心肌细胞膜的钠-钾atp酶泵，导致细胞内的钠和钙的增加，进而增加心肌收缩力。
Digoxin directly inhibits the sodium-potassium ATPase pump in the membrane of the cardiac myocyte, causing an increase in intracellular sodium and calcium which increases myocardial contractility.
地高辛直接抑制心肌细胞膜的钠-钾atp酶泵，导致细胞内的钠和钙的增加，进而增加心肌收缩力。
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