Conclusion Low-frequency ultrasound can induce apoptosis in vitro in C6 cells.
结论：低频超声可以诱导C6细胞凋亡。
Objective to observe the effect of low-frequency ultrasound on apoptosis of C6 cells in vitro.
目的：应用低频超声诱导体外培养C6细胞凋亡，探讨低频超声在体外对肿瘤细胞的影响。
These results indicate that VPA induces a specific differentiation of C6 cells toward a neuronal-like phenotype.
这些结果表明丙戊酸诱导了C6胶质瘤细胞向神经元样显型方向的特异分化。
Objective: To study the function of matrix metalloproteinase-2 (MMP-2) in the selected C6 cells invasion model in vitro.
目的：探讨基质金属蛋白酶2 (MMP - 2)在体外构建的C6细胞侵袭模型中的作用。
The converse effects of B on C6 cells and SK-N-SH cells resulted from different transportation system in two kinds of cells.
皮质酮对C6细胞和SK - N - SH细胞作用的差异可能是由于二种细胞甘氨酸转运系统不同所致。
CONCLUSION: PKC, instead of PKA, was involved in the signal transduction of the rapid effect of B on glycine uptake in C6 cells.
结论：上述结果提示蛋白激酶C参与了硫酸皮质酮快速抑制C6细胞摄取甘氨酸的信号转导过程。
Conclusions SPB can apparently inhibit the proliferation of C6 cells and can induce cell differentiation and even apoptosis of C6 cells.
结论苯丁酸钠可明显抑制C6细胞增殖，诱导细胞分化，甚至诱发细胞凋亡。
Conclusion The changes of microtubule function are involved in glucose uptake by C6 cells and glucose transport process mediated by GLUT1.
结论微管功能变化与C6细胞葡萄糖摄取及葡萄糖转运体介导的葡萄糖转运过程密切相关。
Methods After uranyl acetate staining, the change of ultrastructure of C6 cells before and after PA treatment was observed under transmission el etron microscopy.
方法采用醋酸双氧铀电子染色，电子显微镜（电镜）下观察培养C6细胞在应用苯乙酸后的超微结构变化。
CONCLUSION: the chloride channel blockers NPPB or NFA lessen the oxidative injury of C6 cells through modulating the function of membrane and down-regulating the protein expression of CLIC4.
结论：氯通道阻断剂NPPB或NFA能够在一定程度上减轻氧化应激引起的C6细胞损伤，可能与调节细胞膜功能和下调CLIC 4蛋白表达有关。
C6 cell line and human glioblastoma cells contained 1% cells which were ABCG2 positive.
C6细胞系和人胶质母细胞瘤细胞中均发现有1%左右的细胞ABCG2蛋白表达阳性。
Objective to study the inhibitory effect of methylmercury chloride (MMC) on rat C6 glioma cells in vitro.
目的：通过体外实验探讨氯化甲基汞(MMC)抗大鼠c6胶质瘤细胞的作用。
Objective To study the inhibitory effect of triethyltin chloride(TETC) on the proliferation of cultured rat C6 glioma cells.
目的：探讨氯化三乙基锡（TETC）对体外培养大鼠C6胶质瘤细胞增殖的抑制作用。
Conclusion MMC has cytotoxicity effect on cultured rat C6 glioma cells in vitro, and can inhibit proliferation and induce apoptosis.
结论：MMC能够杀伤大鼠c6胶质瘤细胞，抑制增殖，诱导凋亡，具有应用于胶质瘤治疗的潜在价值。
Cell inhibition rate (IR) was calculated after C6 glioma cells were cultured in the controlled drug release system for testifying tumor-killing effect of the controlled drug release system.
利用C6胶质瘤细胞在药物缓释体中培养后计算细胞抑制率(IR)验证药物缓释体的缓释杀瘤效果。
Objective to explore the changes in apoptosis level and proliferative activity of the C6 brain glioma cells after cryotherapy in rats.
目的探讨大鼠c6脑胶质瘤模型冷冻治疗后细胞凋亡水平和增殖活性的变化。
Objective: To study whether supernatant of C6 glioma cells can induce migration of neural stem cells in vitro.
前言：目的：体外观察c6胶质瘤细胞的培养上清是否有诱导神经干细胞迁移的作用。
Conclusion the Pingliukang medicated serum can inhibit the proliferation and block cell cycle of cultured C6 glioma cells.
结论平瘤康含药血清可能通过阻滞细胞周期而抑制胶质瘤细胞增殖。
Objective: to explore the apoptosis inducing effect of matrine on C6 glioma cells and the related mechanisms.
摘要目的：探讨苦参碱诱导C6胶质瘤细胞凋亡的作用特点和可能的基因作用机制。
Conclusions Lipid-coated microbubbles may carry Taxol to C6 glioma cells, and the compound may act an killing effect in vitro. The mechanisms of metabolism associated with acidic component.
结论脂膜微囊可以承载紫杉醇在体外起到杀伤胶质瘤细胞的作用，且脂膜微囊的代谢与细胞内的酸性成分有关。
Conclusions Lipid-coated microbubbles may carry Taxol to C6 glioma cells, and the compound may act an killing effect in vitro. The mechanisms of metabolism associated with acidic component.
结论脂膜微囊可以承载紫杉醇在体外起到杀伤胶质瘤细胞的作用，且脂膜微囊的代谢与细胞内的酸性成分有关。
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