Pathology. -bronchiolitis is bronchiolar inflammation of various causes.
病理学表现：为各种原因所致的细支气管炎症。
The inflammatory infiltration and hyperplasia in lung tissue, bronchiolar damage and the bronchial mucosa defulvium appeared in the rats exposed to carbonyl nickel.
不同浓度羰基镍染毒大鼠肺组织有炎性渗出和增生，部分细支气管破坏，黏膜坏死脱落；
Stem cells found along the surfaces of the airways (in the bronchiolar epithelium) proliferate rapidly in mice after viral infection and migrate to sites of damage.
在小鼠呼吸道表面(支气管上皮)发现的干细胞在病毒感染后快速增殖并迁移到损伤部位。
Conclusions Long-term smoking may result in the infiltration of inflammatory cells in the bronchiolar walls and the morphology changes of lung tissues, leading to COPD.
结论长期吸烟可引起气道壁炎细胞浸润及肺组织形态学变化，导致COPD发生。
Air trapping secondary to bronchial or bronchiolar obstruction may produce focal zones of decreased attenuation, an appearance that can be enhanced by using expiratory ct.
继发于支气管和细支气管阻塞的空气潴留可产生局部低衰减带，后者应用呼气相ct可增强其表现。
Especially, we have demonstrated S-100 protein in eosinophilic adenoma and medullary carcinoma of thyroid gland, adenocarcinoma of bile duct and bronchiolar adenocarcinoma.
此外，发现支气管腺癌、甲状腺嗜酸性腺瘤、甲状腺髓样癌和胆管细胞癌S-100蛋白阳性。
A common manifestation of bronchiolar edema and stiff lungs due to heart failure is a dry cough, which must be differentiated from that occurring in 5% of patients treated with ACE inhibitors.
心力衰竭引起的细支气管水肿和硬肺常表现为干咳，必须与发生率为5%的ACE抑制剂使用病人的干咳相鉴别。
A common manifestation of bronchiolar edema and stiff lungs due to heart failure is a dry cough, which must be differentiated from that occurring in 5% of patients treated with ACE inhibitors.
心力衰竭引起的细支气管水肿和硬肺常表现为干咳，必须与发生率为5%的ACE抑制剂使用病人的干咳相鉴别。
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