Conclusion: Our results suggest that long-term stimulus with the low-concentration ethanol-induced gastric mucosal cell proliferation is related with BMK1 signaling pathway via ROS production.
结论：慢性饮酒刺激胃黏膜底腺干细胞的增殖可能与酒精引起活性氧（ROS）增多有关，同时本研究还第1次发现慢性饮酒诱导胃底腺干细胞增殖是由BMK1所介导。
Conclusion: Our results suggest that long-term stimulus with the low-concentration ethanol-induced gastric mucosal cell proliferation is related with BMK1 signaling pathway via ROS production.
结论：慢性饮酒刺激胃黏膜底腺干细胞的增殖可能与酒精引起活性氧（ROS）增多有关，同时本研究还第1次发现慢性饮酒诱导胃底腺干细胞增殖是由BMK1所介导。
应用推荐