AQP support, participation and development.
支持、参与和开发AQP。
Conclusion AQP-4 contributed to brain edema formation in the acute brain hemorrhage of human.
结论A Q P - 4参与了人类急性脑出血脑水肿的形成。
AQP-4 deletion aggravated neurological deficits and brain edema contents of whole hemorrhagic ipsilateral hemisphere.
AQP- 4的缺失加剧了神经功能缺损及患侧大脑半球含水量。
Objective To investigate the seropositive rate of AQP-4 in patients of optic neuritis and the effect of diagnosis of recovery and.
目的了解特发性脱髓鞘性视神经炎患者AQP-4的阳性率及对其预后的诊断作用。
Conclusion AQP-1 in CBCEC play an important role in corneal endothelial cell's fluid transport, its expression change may cause corneal edema and function disorder.
结论牛角膜内皮细胞中的水通道蛋白- 1在角膜内皮液体转运中起着重要作用，其表达改变可能导致角膜水肿和功能改变。
CONCLUSION: the expression of AQP-1 in cultured rat pleural mesothelial cells is confirmed at the protein and mRNA level. Dex increases the expression of AQP-1 in a time-dependent manner.
结论：胸膜间皮细胞存在AQP - 1表达，地塞米松对胸膜间皮细胞AQP - 1表达有明显的增强性调节作用，具有时间依赖性。
CONCLUSION: the expression of AQP-1 in cultured rat pleural mesothelial cells is confirmed at the protein and mRNA level. Dex increases the expression of AQP-1 in a time-dependent manner.
结论：胸膜间皮细胞存在AQP - 1表达，地塞米松对胸膜间皮细胞AQP - 1表达有明显的增强性调节作用，具有时间依赖性。
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