Iron plays a very important part in the reactions in our cells that produce damaging free radicals, which glom onto cell membranes and DNA, and may translate into aging the cell.
而铁元素在我们的细胞中产生一种具有破坏性的，能渗透进细胞膜和DNA并老化细胞的物质的过程中扮演着重要的角色。
Free radicals, linked to rapid aging, are highly reactive with other molecules, including vital DNA and proteins, the destruction of which can damage or kill cells.
与迅速衰老相关联的自由基很容易与包括维持生命所必需的DNA和蛋白质发生反应，而破坏这些DNA和蛋白质，就可能会损伤或杀死细胞。
The field of biological aging has in recent years focused on the long molecules of DNA contained in human cells called chromosomes.
在研究生物学老化问题的领域，近年来科学家们把注意力集中在被称作染色体的细胞DNA长分子上。
Scientists identified 150 DNA sequence variations called single nucleotide polymorphisms among those 100 years old and above that may have contributed to their healthy aging.
科学家们辨认了那些百岁或超过百岁老人的150个不同的有助于健康老龄化的被称作单核甘酸多态性的DNA序列。
Here, we review the role of epigenetics in aging and longevity with a focus on DNA methylation.
这里，我们回顾了表观遗传学在衰老和长寿中的作用，集中在DNA甲基化。
This shows the basic reason for skin aging is DNA synthesis obstruction of cells.
这说明DNA合成障碍是皮肤细胞衰老的根本原因。
One line of evidence supporting the connection is that defects in certain genes needed for DNA repair produce maladies that mimic those of accelerated aging.
支持这种关系的证据是需要修复的特定基因的损伤产生疾病例如加速衰老。
The result is yeast cells that have intact DNA but are sterile, a symptom of aging in the fungi.
结果时酵母细胞有了完好的DNA，但是这些细胞不孕了，这是真菌类衰老的症状。
These molecules can react with cells, membranes and DNA, causing skin damage and signs of aging.
这些分子能与细胞，隔膜和DNA起反应，导致皮肤受损并显现衰老迹象。
Outside the cell DNA damage, is effective against free radicals and prevent skin aging.
外线对细胞dna的伤害，有效对抗自由基，防止肌肤老化。
The relation between mutation and DNA damage and cell aging is discussed in this paper.
对降低基因突变和DNA损伤与细胞老化的关系作了讨论。
AIM: To study the effect of medlar on DNA repair after injury, and investigate the pharmacological role of medlar against aging.
目的：检测枸杞子对遗传物质DNA受损后修复的作用，探讨枸杞子在抗衰老中的药理作用。
Objective: The effect of DNA demethylation on aging process and telomere length was studied.
目的：研究DNA甲基化水平变化对细胞衰老进程和端区长度的影响。
Since there was then no DNA programming for aging, those intelligent creatures could live in misery for thousands of your calendar years.
因为没有DNA老化的设计，这些智能生命能够在痛苦中生活数千年。
Cellular aging is linked to deficiencies in efficient repair of DNA double strand breaks and authentic genome maintenance at the chromatin level.
细胞衰老与DNA双螺旋断裂后不能有效修复并在染色质水平维持可靠的基因组有关。
As the DNA damage accumulates, disease and aging set in.
当DNA的损害累积起来时，疾病和衰老便接踵而至。
DNA repair is essential for the accurate preservation of genetic information and to ensure the healthy functioning of cells, and a connection between aging and DNA damage has long been suspected.
DNA修复对遗传信息的准确保存和确保细胞的正常功能是必需的，年龄和DNA损伤的关系一直存在疑问。
Objective to observe the effects of zinc-deficiency (ZD) on antioxidation system and repair of DNA damage and of hepatic cells by establishing an aging mice model induced with D-galactose.
目的通过d半乳糖诱导小鼠衰老模型，探讨缺锌对衰老小鼠抗氧化系统和肝脏dna损伤与修复的影响。
Being the functional fragments of DNA that store and transmit genetic information genes are to result in the aging of cells, diseases and accelerating the process of body's senescence if damaged.
基因是贮存、传递遗传信息的DNA功能片段。基因受损，可导致细胞老化，疾病丛生，加速肌体衰老。
Deacetylation of p53 occurs through interaction with the SIRT1 protein, a deacetylase that may be involved in cellular aging and the DNA damage response (19).
SIRT1蛋白，一种涉及到细胞衰老和DNA损伤反应的去乙酰基酶能够与p53结合，促使后者的去乙酰化（19）。
Deacetylation of p53 occurs through interaction with the SIRT1 protein, a deacetylase that may be involved in cellular aging and the DNA damage response (19).
SIRT1蛋白，一种涉及到细胞衰老和DNA损伤反应的去乙酰基酶能够与p53结合，促使后者的去乙酰化（19）。
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