To explore the adhesion of peripheral blood lymphocytes and eosinophils and lung microvascular endothelial cells (LMEC) in asthma.
观察哮喘豚鼠模型嗜酸细胞和淋巴细胞与肺微血管内皮细胞的粘附率；
Conclusion Intermittent ventilation has the protective effects on lung injury during CPB by decreasing granulocytes adhesion and alleviating lung inflammatory reaction and endothelial cells injury.
结论间断肺通气可通过减少白细胞与血管内皮的黏附，减轻肺部炎症反应、内皮细胞损伤等，对CPB所致的肺损伤有保护作用。
Spontaneous pneumothorax with pleura adhesion can be treated by pneumonolysis and bullae of lung excision under thoracoscopy assisted small incision operation.
自发性气胸并胸膜粘连可用胸腔镜辅助小切口行粘连松解肺大泡切除术；
Conclusion Intrapleural urokinase not only reduces pleural adhesion, loculation and thickening of the pleura, but also improves lung function.
结论胸腔内注射尿激酶能减少胸腔分隔、多房的形成。减少胸膜肥厚，改善肺功能。
CONCLUSION: CCK-8 attenuates LPS-induced acute lung injury by means of anti-oxidation and inhibition of PMN adhesion and aggregation, both of which are mediated by endogenous H2S.
结论：CCK- 8可通过内源性H2S介导的抗氧化、抑制PMN黏附聚集等效应发挥减轻LPS所致肺损伤的作用。
Objective: to observe the MODS rat lung tissue's CEC, as well as adhesion molecules ICAM-1 and VCAM-1 expression changes, and to investigate the relationship between them.
目的：观察多器官功能障碍综合征(MODS)大鼠肺组织中循环内皮细胞(CEC)以及细胞间黏附分子(ICAM - 1)和血管细胞黏附分子(VCAM - 1)的表达，并探讨它们之间的相互关系。
To investigate the quantitative expression and roles of protein kinase C (PKC), Variant CD44 (CD44V3-10) adhesion molecules in Non-small cellular lung cancer (NSCLC).
目的研究蛋白激酶C（PKC）、粘附分子CD44变构体CD44V3-10在非小细胞性肺癌（NSCLC）中定量表达及在肺癌发生及转移中的作用。
To investigate the quantitative expression and roles of protein kinase C (PKC), Variant CD44 (CD44V3-10) adhesion molecules in Non-small cellular lung cancer (NSCLC).
目的研究蛋白激酶C（PKC）、粘附分子CD44变构体CD44V3-10在非小细胞性肺癌（NSCLC）中定量表达及在肺癌发生及转移中的作用。
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