CONCLUSION: The expression levels of ADAM 10 and ADAM 17 in hippocampus of old men may not be a part of biologic foundation of Alzheimer disease.

结论：去整合蛋白和金属蛋白水解酶10和17的基因表达水平可能不是老龄人易发阿尔茨海默病的生物学基础。

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CONCLUSION: The expression levels of ADAM 10 and ADAM 17 in hippocampus of old men may not be a part of biologic foundation of Alzheimer disease.

结论：去整合蛋白和金属蛋白水解酶10和17的基因表达水平可能不是老龄人易发阿尔茨海默病的生物学基础。

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