目的研究外源性单唾液酸四己糖神经节苷脂（GM1）对新生鼠缺氧缺血脑损伤（HIBD）的保护作用。
AIM To study the protective effects of monosialotetrahexosylganglioside (GM1) after hypoxic ischemic brain damage (HIBD) in neonatal rats.
通过亚低温对新生鼠缺氧缺血性脑损伤（HIBD）大脑皮质神经元一氧化氮合酶（NOS）及血糖水平影响的研究，探讨亚低温对缺氧缺血性脑损伤的保护作用机制。
Through investigating the effect of mild hypothermia on activity of nitric oxide synthase(NOS) in cortical neurons and glycemia levels of neonatal rats with hypoxic ischemic brain damage(HIBD).
目的了解新生鼠缺氧缺血性脑损伤(HIBD)时大脑皮质神经元特异性烯醇化酶(nse)和胶质纤维酸性蛋白(GFAP)的变化。
Objective To observe the change of neuron-specific enolase (NSE) and glial fibrillary acidic protein (GFAP) in neonatal rats' cerebral cortex with hypoxic-ischemic brain damage (HIBD).
结论：NOS抑制剂通过减轻缺血缺氧脑损伤时胎鼠脑神经细胞凋亡对其出生后的远期智能起保护作用。
Conclusion: NOS inhibitors play a obvious protective role in long-term intelligence of fetal rat after delivery through relieved neuron apoptosis during hypoxic ischemia encephalopathy.
目的探讨16二磷酸果糖(FDP)干预治疗新生鼠缺氧缺血性脑损伤(HIBD)后脑神经细胞线粒体形态学变化及对能量代谢的影响。
Objective to study the morphologic changes of mitochondria and metabolic protective effect of fructose 1 6 diphosphate (FDP) intervention in newborn rats with hypoxic ischemic brain damage (HIBD).
目的探讨16二磷酸果糖(FDP)干预治疗新生鼠缺氧缺血性脑损伤(HIBD)后脑神经细胞线粒体形态学变化及对能量代谢的影响。
Objective to study the morphologic changes of mitochondria and metabolic protective effect of fructose 1 6 diphosphate (FDP) intervention in newborn rats with hypoxic ischemic brain damage (HIBD).
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