目的探讨饮食诱导肥胖抵抗(DIO - R)大鼠和饮食诱导肥胖(DIO)大鼠的肥胖相关指标的变化。
Objective To study the change of obesity related index in diet-induced obesity-resistant (DIO-R) rats and diet-induced obesity (DIO) rats.
实验结束时,根据体重将高脂实验组大鼠分为饮食诱导肥胖(DIO)和饮食诱导肥胖抵抗(DIO R)大鼠,比较各组相关指标的差异。
At the end of the experiment, the high-fat diet group was divided into dietary induced obesity (DIO) and dietary induced obesity resistant (DIO-R) subgroups according to the final body weight.
同一种系大鼠中存在饮食诱导肥胖和胰岛素抵抗的个体差异,肥胖抵抗大鼠不易发生胰岛素抵抗。
There was difference of fat rich diet induced obesity and insulin resistance in the same species rats, the obesity prone rats had more tendency to become insulin resistance.
结论含脂量38.9%的高脂饮食可诱导大鼠出现肥胖、高胰岛素血症、血脂异常等胰岛素抵抗综合征的表现。
Conclusion fat rich diet (fat content 38.9%) can induce rats the characteristic of insulin resistance syndrome such as obese, hyperinsulinemia, dyslipidemia etc.
目的探讨高脂饮食诱导肥胖易感(OP)大鼠和肥胖抵抗(OR)大鼠,激素敏感性脂肪酶(HSL)和脂蛋白脂肪酶(LPL)基因表达的差别。
Objective To explore the gene expression of hormone sensitive lipase (HSL) and lipoprotein lipase (LPL) between obesity-prone (op) and obesity-resistant (or) rats induced by a high-fat diet.
目的探讨解偶联蛋白在大鼠抵抗饮食诱导肥胖中的作用。
Objective To explore the role of uncoupling proteins (UCP) in resisting diet-induced obesity in rats.
方法:建立高脂饮食诱导的肥胖、胰岛素抵抗大鼠模型,检测大鼠体重、空腹胰岛素、空腹血糖及血脂水平。
Methods the fat rat models were developed by given oil-rich food, and the body weight, fasting insulin, fasting blood sugar and blood lipid of the models were measured.
在同一种系大鼠中存在饮食诱导肥胖和胰岛素抵抗的个体差异,肥胖抵抗大鼠不易发生胰岛素抵抗。
There was difference of fat - rich - diet induced obesity and insulin resistance in the same species species rats, the obesity - prone rats had more tendency to become insulin resistance.
在同一种系大鼠中存在饮食诱导肥胖和胰岛素抵抗的个体差异,肥胖抵抗大鼠不易发生胰岛素抵抗。
There was difference of fat - rich - diet induced obesity and insulin resistance in the same species species rats, the obesity - prone rats had more tendency to become insulin resistance.
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