同一种系大鼠中存在饮食诱导肥胖和胰岛素抵抗的个体差异，肥胖抵抗大鼠不易发生胰岛素抵抗。
There was difference of fat rich diet induced obesity and insulin resistance in the same species rats, the obesity prone rats had more tendency to become insulin resistance.
结论含脂量38.9%的高脂饮食可诱导大鼠出现肥胖、高胰岛素血症、血脂异常等胰岛素抵抗综合征的表现。
Conclusion fat rich diet (fat content 38.9%) can induce rats the characteristic of insulin resistance syndrome such as obese, hyperinsulinemia, dyslipidemia etc.
目的探讨解偶联蛋白在大鼠抵抗饮食诱导肥胖中的作用。
Objective To explore the role of uncoupling proteins (UCP) in resisting diet-induced obesity in rats.
目的探讨高脂饮食诱导肥胖易感(OP)大鼠和肥胖抵抗(OR)大鼠，激素敏感性脂肪酶(HSL)和脂蛋白脂肪酶(LPL)基因表达的差别。
Objective To explore the gene expression of hormone sensitive lipase (HSL) and lipoprotein lipase (LPL) between obesity-prone (op) and obesity-resistant (or) rats induced by a high-fat diet.
在同一种系大鼠中存在饮食诱导肥胖和胰岛素抵抗的个体差异，肥胖抵抗大鼠不易发生胰岛素抵抗。
There was difference of fat - rich - diet induced obesity and insulin resistance in the same species species rats, the obesity - prone rats had more tendency to become insulin resistance.
目的探讨饮食诱导肥胖抵抗(DIO - R)大鼠和饮食诱导肥胖(DIO)大鼠的肥胖相关指标的变化。
Objective To study the change of obesity related index in diet-induced obesity-resistant (DIO-R) rats and diet-induced obesity (DIO) rats.
方法：建立高脂饮食诱导的肥胖、胰岛素抵抗大鼠模型，检测大鼠体重、空腹胰岛素、空腹血糖及血脂水平。
Methods the fat rat models were developed by given oil-rich food, and the body weight, fasting insulin, fasting blood sugar and blood lipid of the models were measured.
方法：建立高脂饮食诱导的肥胖、胰岛素抵抗大鼠模型，检测大鼠体重、空腹胰岛素、空腹血糖及血脂水平。
Methods the fat rat models were developed by given oil-rich food, and the body weight, fasting insulin, fasting blood sugar and blood lipid of the models were measured.
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