结果和结论：吸入性伊洛前列素治疗可以降低肺血管阻力和延缓原发性肺动脉高压的进程。
Results and Conclusion: Inhaled iloprost can reduce pulmonary vascular resistance and delay the development of primary pulmonary hypertension.
肺缺血再灌注损伤可以表现为肺血管阻力升高，氧合功能下降，顺应性降低以及水肿形成等等。
The manifestations of pulmonary ischemia -reperfusion injury can be characterized by increased pulmonary vascular resistance, decreased oxygenation capacity, worsened compliance, and edema formation.
急性肺栓塞使肺动脉压力及肺血管阻力明显升高，溶栓疗法使肺动脉压力及肺血管阻力明显降低，对照组两参数在栓塞后随时间无明显变化。
Pulmonary artery pressure and pulmonary resistance increased markedly after pulmonary embolism, which were lowered progressively and significant by thrombolytic therapy in contrary with control dogs.
急性肺栓塞使肺动脉压力及肺血管阻力明显升高，溶栓疗法使肺动脉压力及肺血管阻力明显降低，对照组两参数在栓塞后随时间无明显变化。
Pulmonary artery pressure and pulmonary resistance increased markedly after pulmonary embolism, which were lowered progressively and significant by thrombolytic therapy in contrary with control dogs.
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