结论哮喘组大鼠肺组织活性氧产量增加。MT干预可以降低肺组织活性氧产生,降低气道炎症和气道高反应性,这可能是其治疗哮喘的保护机制。
Conclusions MT can decrease airway inflammation and the content of ROS in asthmatic rats, which may be the underlying protective mechanisms of MT against asthma.
结论哮喘组大鼠肺组织活性氧产量增加。MT干预可以降低肺组织活性氧产生,降低气道炎症和气道高反应性,这可能是其治疗哮喘的保护机制。
Conclusions MT can decrease airway inflammation and the content of ROS in asthmatic rats, which may be the underlying protective mechanisms of MT against asthma.
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