接受亚铁血红素后，1型糖尿病大鼠的内皮形态与对照组相似。
The endothelial morphology in type 1 diabetic rats was similar to that of the control group after receiving ferriheme.
非亚铁血红素和癌症之间没有联系。
清单10中的结果显示了铁经常与亚铁血红素化合物共同出现。
The result in Listing 10 shows that iron occurs frequently together with heme compounds.
目的研究微粉化工艺对羊血提取物中氯高铁血红素溶出度的影响。
Objective To study the effect of micronization technology on the dissolution of hemin from the extract of sheep blood.
目的探讨正铁血红素对急性间歇性血卟啉症(aip)的治疗作用。
Aim To evaluate the therapeutic efficacy of Panhematin for acute intermittent porphyria (AIP).
氢氧自由基可能是抗坏血酸增强氯高铁血红素诱导红细胞溶血的原因；
HO might be the cause of ascorbic acid-enhanced hemolysis induced by hemin.
目的研究氯高铁血红素对慢性肾衰竭大鼠血压的影响，并探讨其可能机制。
To study the effect of hemin on nephrogenic hypertension of chronic renal failure rats and its possible mechanism .
抗坏血酸在氯高铁血红素存在时，可以作为一个亲氧化剂而非一个抗氧化剂。
Ascorbic acid could function as a prooxidant, rather than an antioxidant, in the presence of hemin.
研究小组发现，线粒体膜上abcb 6蛋白的增加导致细胞产生更多的亚铁血红素必须的比咯紫质。
The team found that the increase in ABCB6 in mitochondrial membranes caused the cells to make more porphyrin, the building block of heme.
还研究了青蒿素与氯高铁血红素的作用情况，发现两者作用后，明显改变了青蒿素在银表面上的吸附。
The interaction between artemisinin and hemin was also studied using SERS spectra. The adsorption of artemisinin on silver had evident change after addition of hemin.
目的：研究血红素氧化酶1的诱导剂高铁血红素在对抗大鼠心肌缺血-复灌损伤中的作用及其相应机制。
Objective: To investigate the effects of heme oxygenase 1 inducer hemin on protection of ischemia-reperfusion injury in rats and its mechanisms.
目的研究可水解单宁诃子酸和特里马素I对氯化高铁血红素和丁酸钠诱导K562细胞红系分化的影响。
Objective Effects of plant tannins, chebulinic acid and tellimagrandin I on chemically induced hemoglobin synthesis in K562 cells were investigated.
亚铁血红素 (HEM)是一个多原子、非蛋白质的有机结构，能够将一个铁 (FE)离子置于其中心。
A heme (HEM) is a multi-atom, non-proteinaceous organic structure capable of positioning an iron (FE) ion in its center.
我们提出了肺结核分支杆菌细胞色素P 450CYP121与三氮唑类药物氟康唑的复合物的晶体结构，揭示了唑类与亚铁血红素新的结合模式。
We present here the crystal structure of Mycobacterium tuberculosis cytochrome P450 CYP121 in complex with the triazole drug fluconazole, revealing a new azole heme ligation mode.
氢氧自由基淬灭剂显著地抑制抗坏血酸增强氯高铁血红素诱导红细胞溶血。由上述可得到如下结论：抗坏血酸增加氯高铁血红素诱导的红细胞氧化压力与H2O2有关；
Several HO. scavengers significantly inhibited hemo lysis induced by hemin and ascorbic acid. It was concluded that H2O2 was involved in the oxidative stress mediated by hemin and ascorbic acd.
铁元素从血红素中释放出来，要么转化为巨噬细胞中的铁蛋白或含铁血黄素，或通过血液循环回流进入骨髓。
Iron is released from the heme moiety and either stored in the macrophage as ferritin or hemosiderin, or released into the circulation for transport back to the marrow.
在NF 3暴露停止后，高铁血红蛋白转化回血红素。高铁血红蛋白症会在几个小时后自动消失，而溶血性贫血需要几个星期才能解除。
At the cessation of NF3exposure, methemoglobin reverts back to hemoglobin. While methemoglobinemia clears spontaneously over several hours, hemolytic anemia may take several weeks to resolve.
在NF 3暴露停止后，高铁血红蛋白转化回血红素。
At the cessation of NF3exposure, methemoglobin reverts back to hemoglobin.
在NF 3暴露停止后，高铁血红蛋白转化回血红素。
At the cessation of NF3exposure, methemoglobin reverts back to hemoglobin.
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