钙超载是心肌顿抑发生的重要机制。
Calcium overloading has been identified as an important pathogenetic mechanism in myocardial stunning.
心肌缺血再灌注损伤的主要机制为氧自由基大量释放，细胞内钙超载以及缺血导致的大量儿茶酚胺释放。
The mechanism of myocardium ischemia reperfusion injury involve in generating of reactive oxygen species, overload of Ca2 + and releasing of catecholamine by ischemia.
目的：研究蕨麻正丁醇部位对大鼠心肌细胞缺氧所致钙超载的抑制作用，并探讨其可能的机制。
Objectives: To study the inhibition and mechanisms of n-butanol fraction of Potentilla anserine L. against hypoxia -induced calcium overload in myocardial cells.
目的：研究蕨麻正丁醇部位对大鼠心肌细胞缺氧所致钙超载的抑制作用，并探讨其可能的机制。
Objectives: To study the inhibition and mechanisms of n-butanol fraction of Potentilla anserine L. against hypoxia -induced calcium overload in myocardial cells.
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