结论福辛普利不仅能防治心肌肥厚，而且能保护肌浆网钙泵功能，防止心肌钙超载损伤。
Conclusions Fosinopril could not only prevent and treat myocardial hypertrophy, but also could maintain the functions of SERCA and prevent myocardial calcium overload.
钙超载是心肌缺血再灌注不可逆损伤的最后通路，但其具体作用途径有待于进一步研究。
Calcium overload is the final pathway of myocardial ischemia reperfusion injury, but the concrete mechanism needs further research.
结论：健心合剂对大鼠心肌缺血损伤具有保护作用，其机理可能与钙超载有关。
Conclusion: Jianxin mixture has a protective effect on myocardial ischemia injury in the rats, which is due to its inhibition of calcium overload.
结论：老龄大鼠脑缺血再灌注脑组织损伤与钙超载和自由基损伤有关。
Conclusion: the calcium overload and free radical injury were correlated with brain ischemia reperfusion injury in the aged rats.
心肌缺血再灌注损伤的主要机制为氧自由基大量释放，细胞内钙超载以及缺血导致的大量儿茶酚胺释放。
The mechanism of myocardium ischemia reperfusion injury involve in generating of reactive oxygen species, overload of Ca2 + and releasing of catecholamine by ischemia.
结果超生理剂量地塞米松可抑制骨骼肌细胞的增殖，降低其SOD水平，提高MDA水平，并造成细胞的损伤与钙超载。
Results Dexamethasone beyond physical dosage could suppress cells proliferation, reduce SOD level, increase MDA, and cause cells injury and Ca 2+ overloading.
视网膜缺血再灌注损伤是多因素综合作用的结果，主要包括氧自由基的损伤作用，细胞内的钙超载现象，白细胞作用以及细胞凋亡等。
Multiple factors cause retinal ischemia-reperfusion injury, including the injury effect of oxygen-derived free radicals, intracellular calcium overload, the action of leucocyte and apoptosis.
视网膜缺血再灌注损伤是多因素综合作用的结果，主要包括氧自由基的损伤作用，细胞内的钙超载现象，白细胞作用以及细胞凋亡等。
Multiple factors cause retinal ischemia-reperfusion injury, including the injury effect of oxygen-derived free radicals, intracellular calcium overload, the action of leucocyte and apoptosis.
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