• 患有Hunter综合征缺乏艾杜糖醛酸2-硫酸酯对于回收复杂水化合物至关重要。

    People with Hunter syndrome lack the enzyme iduronate-2-sulfatase, which is essential for recycling complex carbohydrates.

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  • 举例来说,不症不是免疫系统过分热情引起的,而是缺乏一种结果身体需要这种分解牛奶主要——乳糖

    Rather than being caused by an overenthusiastic immune system, milk intolerance, for example, results from a lack of the enzyme the body needs to break down lactose, the main sugar in milk.

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  • 虽然容易用数学建模,但实验室实现这个系统让人颇费思量,因为里面缺乏帮助组装单体模板

    Although it is easy to model mathematically, making the system in the lab is tricky because it starts without any enzymes or templates to help the monomers assemble.

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  • 知道,亚贡雪莲果的菊糖是直接通过消化区域而不用产生代谢变化的,这适合那些缺乏必需代谢的胰岛素食用。

    You see, the human body lacks the enzyme necessary to metabolize insulin, so the substance that gives yacon its sweetness passes through the digestive tract unmetabolized.

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  • 酵解关键——丙酮,如果它有遗传缺陷,就导致会atp缺乏红细胞寿命缩短溶血性贫血

    Inherited deficiency of pyruvate kinase, a key glycolytic enzyme, causes ATP deficiency, which leads to reduced RBC life span and hemolytic anemia.

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  • 左图野生斑马鱼胚胎染上红色血细胞右图缺乏Ago2变异胚胎则不能产生血细胞

    On left, red blood cells are stained in wild type zebrafish embryos while on the right mutant embryos lacking the enzyme Ago2 fail to produce blood cells.

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  • 黑色素细胞形态都是正常的,只是先天性氨酸减少缺乏致使不能产生黑色素所引起的。

    Melanoma cells and its patterns are normal, but congenital enzyme tyrosine sharp reduction or lack of, with the result that should not have caused melanoma.

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  • 他们可能现在任何一活着细胞简单也许缺乏蛋白质催化也没有我们熟悉具磷酸核糖股价的遗传大分子

    They were probably simpler than any cell now alive, and may have lacked not only protein-based catalysis, but perhaps even the familiar genetic macromolecules, with their ribose-phosphate backbones.

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  • 一部份原因缺乏蛋白质分解制造结构

    A part of the reason for this is a lack of enzymes to break down the older proteins to be recreated into the new structure.

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  • HBV复制过程中要经过逆转录,由于逆转录缺乏有效碱基校对功能hbv基因组一般dna病毒发生变异。

    As the reverse transcriptase is lack of effective function of proofreading the bases, the HBV genome is easier to mutate than other DNA viruses.

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  • 英国内科医生。他首先发现一类新的基因诱导疾病是由于某一个基因发生突变从而导致某种特殊缺乏产生故障而引发的。

    British physician who first recognized a new class of genetically induced diseases resulting from a mutation in a single gene that causes the absence or malfunctioning of a particular enzyme.

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  • 这些结果提示具有良好营养状态机体运动应激提高能量代谢活性适应机体的需要缺乏机体对运动应激不能作出适应性改变。

    These results suggested that the body in good se status could raise the enzyme activity of energy metabolism in exercise to meet the body's need, while the body in poor se condition could not.

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  • 一个X染色体缺陷导致尿素循环缺乏之一

    This is an X-linked deficiency of one of the enzymes in the urea cycle.

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  • 目的探讨葡萄糖6磷酸脱氢缺乏程度患儿高胆红素血症发生率关系

    Objective: To evaluate the relationship between the incidence of hyperbilirubinemia and the degree of the G6PD (glucose-6 phosphate dehydrogenate) deficiency.

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  • 目的从红细胞膜蛋白磷酸化改变角度探讨葡萄糖6磷酸脱氢(G6PD)缺乏溶血机制

    Objective To explore the hemolytic mechanism of glucose 6 phosphate dehydrogenase (G6PD) deficient erythrocytes in the view of phosphorylation of membrane protein.

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  • 目的阐明海南汉族黎族人群葡萄糖-6-磷酸脱氢酶缺乏分子基础

    Objective:To elucidate the molecular basis of G6PD deficiency in the Han and Li nationalities in Hainan, China.

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  • 目的建立适于葡萄糖6磷酸脱氢(G6PD)缺乏新生儿筛查检测方法

    Objective To establish the methods for neonatal screening of glucose 6 phosphate dehydrogenase (G6PD) deficiency.

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  • 具革新意义的经典18 -糖基-水解家族包括缺乏水解活性类壳质蛋白物

    Background The evolutionarily conserved 18-glycosyl-hydrolase family contains true chitinases and chitinase-like proteins that lack enzymatic activity.

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  • 目的建立适合葡萄糖6-磷酸脱氢(G6PD)缺乏新生儿筛查检测方法

    Objective To establish the methods for neonatal screening of glucose-6 phosphate dehydrogenase (G6PD) deficiency.

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  • 90%人类肿瘤可检测到活性,正常体细胞缺乏这种活性

    Telomerase activity has been detected in almost 90%of all human cancers, but is generally absent in normal somatic cells.

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  • 目的研究葡萄糖6磷酸脱氢(G6PD)缺乏新生儿高胆红素血症发生率及发病特点

    Objective to research into the morbidity and characteristics of hyperbilirubinemia which develops among the G6PD (glucose-6 phosphate dehydrogenate) deficiency neonates.

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  • 那就是肿瘤细胞可能缺乏一个名为TCPTP关键

    They may lack a key enzyme called TCPTP.

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  • 蚕蛾溶茧可能来源于下颚、嗉囊和中,嗉囊是否具有分泌溶茧能力至今缺乏直接证据

    The origin of cocoonase of silkmoth, Bombyx mori was likely midgut, maxilla, or craw. However, it was absent of direct evidences whether the craw could secret cocoonase .

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  • 目的:测定葡萄糖- 6 -磷酸脱氢(G6PD)缺乏时红细胞膜唾液含量进一步了解G6PD缺乏溶血的机理。

    Objective: the erythrocyte membrane contents of sialic acid was estimated in order to study the Glucose-6-phosphate dehydrogenase (G6PD) deficient erythrocyte hemolysis.

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  • 结论缺乏较为严重地影响奶牛红细胞组织atp活性抑制了这些组织的正常生理功能

    Conclusion: Copper deficiency had severe effect on the activities of the membrane ATPase of red cell and tissue in calves and restrained the normal physiological function of these tissue.

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  • 葡萄糖- 6 -磷酸脱氢(G6PD)缺乏常见遗传性病,主要由于G6PD基因突变所致。

    Glucose-6-phosphate dehydrogenase (G6PD) deficiency is the most frequent genetic enzymopathy, mainly caused by mutations of G6PD gene.

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  • 葡萄糖- 6 -磷酸脱氢(G6PD)缺乏常见遗传性病,主要由于G6PD基因突变所致。

    Glucose-6-phosphate dehydrogenase (G6PD) deficiency is the most frequent genetic enzymopathy, mainly caused by mutations of G6PD gene.

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