结论急性白血病存在造血微环境异常。
Conclusion There were many abnormalities in hematopoietic inductive microenvironment (HIM) of acute leukemia.
目的探讨放烧复合伤对骨髓造血微环境损伤的机制。
Aim to investigate the mechanism of the radiation burn combined injury to the bone marrow hematopoietic microenvironment in the mice.
目的探讨人急性白血病骨髓造血微环境与正常的差异。
Objective to study the difference of bone marrow hematopoietic microenvironment between acute leukemia and normal controls.
减轻骨髓微血管损害，促进辐射后造血微环境的恢复；
双黄升白冲剂对小鼠骨髓有良好的保护、改善造血微环境的作用。
Experimental study showed that SHSBG has good bone marrow hematopoietic microenvironment protecting and improving effect in mice.
目的：进一步探讨外周血干细胞移植后患者骨髓造血微环境损伤的机理。
Objective: to investigate the injury mechanism of bone marrow hematopoietic microenvironment from PBSCT patients anterior and post pretreatment.
摘要骨髓造血微环境存在于全身骨骼的骨髓腔内，是机体造血以及免疫细胞发育的场所。
Bone marrow hematopoietic microenvironment occupies the medullary cavities of bones throughout the skeleton and provides support for hematopoiesis and immune cells development.
实验部分通过兔疫介导的再障小鼠骨髓基质细胞体外培养探索活髓片在骨髓造血微环境方面的作用机理。
In laboratory research, we observed effect of Huo Shui TAB on the bone marrow micro-environment of immune-mediated aplastic anemia on mice by in vitro culture of bone marrow stromal cell.
目的： 用间质干细胞（MSC）体外模拟造血微环境，探讨脐带血造血前体细胞在MSC微环境中的增殖和分化特性。
AIM: To investigate the proliferation and differentiation patterns of hematopoietic precursors from cord blood in mesenchymal stem cell(MSC) microenvironment.
关于造血功能损伤与修复的研究与应用，既往多侧重于造血实质细胞，对构成造血微环境重要成分的造血基质细胞尚需深入研究。
The research and application on injury and repair of hematopoietic function in the past focus more on HSC, and it is needed to further study of the stromal cells.
在CFU - F减低组中循环免疫复合物(CIC)升高的病例不比CFU - F正常组中CIC升高的病例明显增多，未能说明CIC升高和造血微环境缺陷有关。
Circulating immune complex (CIC) elevated cases in CFU-F reduced group were not more common than that in CFU-F normal group. Thus the elevation of CIC was not related to micro-environment defect.
在CFU - F减低组中循环免疫复合物(CIC)升高的病例不比CFU - F正常组中CIC升高的病例明显增多，未能说明CIC升高和造血微环境缺陷有关。
Circulating immune complex (CIC) elevated cases in CFU-F reduced group were not more common than that in CFU-F normal group. Thus the elevation of CIC was not related to micro-environment defect.
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