结论低潮气量通气治疗ARDS动物模型能够减轻炎症诱发细胞因子的释放，减轻机械通气相关性肺损伤。

Conclusion Low tidal volume ventilation can decrease cytokine release and can alleviate ventilator - induced lung injury (VILI) in animal models of ARDS.

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目的探讨允许性高碳酸血症肺保护性通气策略对降低呼吸窘迫综合征新生儿(NRDS)呼吸机相关性肺损伤的价值。

Objective to investigate the advantages of protective ventilation strategies in NRDS and the value of decreasing the incidence of ventilator-associated lung injury (VALI).

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目的通过建立过度通气导致呼吸机相关性肺损伤(VILI)大鼠模型，评估不同潮气量对肺损伤的影响。

Objective to establish an experimental model of ventilator-induced lung injury (VILI) in rats through tracheal tube and evaluate the influence of different ventilatory volumes on VILI in rat lung.

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目的通过建立过度通气导致呼吸机相关性肺损伤(VILI)大鼠模型，评估不同潮气量对肺损伤的影响。

Objective to establish an experimental model of ventilator-induced lung injury (VILI) in rats through tracheal tube and evaluate the influence of different ventilatory volumes on VILI in rat lung.

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