目的：研究运动性心肌肥大形成中血浆生物活性肽的含量及意义。
Objective:To investigate the changes of plasma biological active peptides in exercise-induced cardiac hypertrophy in rats.
本研究提示运动性心肌肥大的心肌抗缺血再灌注损伤的能力较高血压心肌肥大者增强。
It suggested that the ability of anti-injury in exercise-induced cardiac hypertrophy is higher than that in hypertension.
运动心脏学已成为一门新兴的交叉学科，本文就运动心脏学的基础研究、运动性心肌肥大；
Sports Cardiology has become anew overlapping school subject is paper presents a discussion about basic research on Sports Cardiology, motional myocardial Hypertrophy;
目前，对运动性心肌肥大属于调节性、生理性肥大的认识渐趋一致，但其发生机制尚未完全阐明。
At present , the recognization is agreed on that cardiac hypertrophy belongs to regulatory and physical cardiac enlargement. But its mechanisms are still not be stated clearly.
仅从运动性心肌肥大的生物学机制方面，就诱导其发生的刺激因素及其信号转导通路、基因表达等几个方面加以综述。
In this article we only reviewed the biological mechanism of exercise induced cardiac hypertrophy by its inductive stimulus, pass way for signal transduction and genetic expression.
资料综合：在持续性运动期间运动诱导心肌肥大是心输出量增加的生理基础。
DATA SYNTHESIS: During continuous exercise, myocardium hypertrophy induced by exercise was the physical basis of the increase in cardiac output.
资料综合：在持续性运动期间运动诱导心肌肥大是心输出量增加的生理基础。
DATA SYNTHESIS: During continuous exercise, myocardium hypertrophy induced by exercise was the physical basis of the increase in cardiac output.
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