操弄辣椒素受体的做法还不仅于此。
The possibilities for manipulating the receptor do not end there.
因此，能够抑制辣椒素受体的物质，应该就可以减轻由发炎引起的疼痛。
Substances that inhibit the capsaicin receptors should therefore dampen inflammatory pain.
第三章致力于吲唑脲类辣椒素受体（TRPV1）通道拮抗剂的定量构效关系（QSAR）研究。
The third chapter was devoted to the study of quantitative structure-activity relationship(QSAR) for indazolyl ureas as TRPV1 antagonists.
目的：通过调节辣椒素受体1 (VR1)基因表达，对VR 1基因的功能及疼痛的基因治疗进行基础研究。
Objective: By regulation of VR1 gene expression, fundamentally investigate function of VR1gene and pains gene therapy.
方法采用全细胞膜片钳技术在急性分离的大鼠背根神经节细胞上观察龙血素B对辣椒素诱发的辣椒素受体电流的影响。
Methods In acutely isolated rat DRG neurons, effects of loureirin B on capsaicin-evoked currents were observed using whole-cell patch-clamp technique.
但通过允许它经TRPV1通道（一种辣椒素受体）进入细胞可保证其止痛特效，这个通道在细胞中只在痛觉神经元中表达。
But antipain specificity is ensured by allowing it to enter via the TRPV1 channel, a capsaicin receptor found only in pain-sensing neurons.
辣椒素是香草酸(vanilloid)分子家族的成员之一，它会与舌头上香草酸受体1亚型(vanilloidreceptor subtype 1)结合。
Since capsaicin is a member of the vanilloid family of molecules, it binds to a receptor on the tongue called the vanilloid receptor subtype 1.
但是奇怪的是VR1受体并不是专门用来检测辣椒素的，它们只和辣椒素结合只是偶然发生的。
But here's the strange part: VR1 receptors weren't designed to detect capsaicin. They bind spicy food by accident.
当辣椒素和这些受体结合后，感觉神经元失去电子，并传递辛辣刺激的信号。
After capsaicin binds to these receptors, the sensory neuron is depolarized, and it sends along a signal indicating the presence of spicy stimuli.
当辣椒素和这些受体结合后，感觉神经元失去电子，并传递辛辣刺激的信号。
After capsaicin binds to these receptors, the sensory neuron is depolarized, and it sends along a signal indicating the presence of spicy stimuli.
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