据报道，突变的p 53可以与其他转录因子相互作用，并且被招募到它们的结合位点以调节它们靶基因的表达。
It has been reported that mutant p53 can interact with other transcription factors and be recruited to their binding sites to modulate the expression of their target genes.
研究信号转导途径中蛋白与蛋白之间或者转录因子之间相互作用对于掌握调控机制和分子组成以及揭示生命活动现象的本质有重要意义。
Studies on interactions of protein-protein or transcription will be important to understand the function and constitute of signaling pathway, and the native characteristics of life.
已有研究表明DNA顺式作用元件与反式作用因子之间的相互作用是染色质重塑和基因转录调节的基础。
Present studies indicate that the interactions between trans-acting factors and corresponding DNA cis-acting elements are the basis of chromatin remodeling and gene transcription regulation.
结论：能方便地获得TTF1体外翻译蛋白，为进一步研究TTF1蛋白与相应的DNA反应元件及其他转录因子的相互作用奠定了基础。
Conclusion: Expression of TTF1 in vitro can be used in the further research of interaction of TTF1 and its DNA response elements and others transcription factors.
进一步工作利用酵母单杂交系统，寻找与-130EBS元件相互作用的转录因子。
Moreover, yeast one-hybrid screen was performed in search of the factors binding the -130EBS element.
DNA甲基化由DNA甲基转移酶催化，在多种调节因子的参与下，与组蛋白修饰相互作用，抑制基因转录，导致基因沉默。
DNA methylation catalyzed by DNA methyltransferases interacts with histone modification to inhibit gene transcription, induce gene silencing at the participation of many regulators.
转录因子与DNA的相互作用是基因表达调控系统的重要组成部分。
The interaction between transcription factors and their DNA recognition sites are important for the regulation of the network that controls gene expression.
AR与BA的发病机制至今仍不十分清楚，其是涉及多种炎性细胞的相互作用，诸多炎性介质、细胞因子、转录因子等共同参与的慢性炎症反应。
The pathogenesis of AR and BA has not been quite clearly, it may be involved in the interaction of many inflammatory mediators, cytokines and transcriptional factors.
这种独一无二的特性受到来自于转录因子和表观遗传调控因子二者间复杂相互作用的调控。
This unique property is controlled by a complex interplay between transcriptional factors and epigenetic regulators.
特别地，转录被转录因子和它们相应的结合位点的相互作用所调控。
In particular, transcription is modulated by the interaction of transcription factors with their corresponding binding sites.
细胞核中，抑制素通过与转录因子如rb和p 53相互作用来调节靶基因的转录(2,5)。
In the nucleus prohibitins interact with transcription factors such as Rb and p53 to regulate target gene transcription (2, 5).
细胞核中，抑制素通过与转录因子如rb和p 53相互作用来调节靶基因的转录(2,5)。
In the nucleus prohibitins interact with transcription factors such as Rb and p53 to regulate target gene transcription (2, 5).
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