素-6（IL-6）在新生儿败血症及败血症休克中的变化及临床意义。
Objective To investigate the clinic value of interleukin-6 (IL-6) in sepsis and septic shock.
目的：观察败血症休克大鼠主动脉内膜、中膜和外膜一氧化氮合成途径的改变。
AIM: To observe the change of nitric oxide (NO) generation system in the vascular adventitia, media and intima in septic shock rats.
实验表明败血症休克时低血压的发生与血管平滑肌细胞HO活性增加和内源性CO生成增多明显相关；
These findings suggest that HO activity and CO release within vascular musculature are increased during septic shock;
目的观察两种败血症休克模型大鼠的血流动力学及心肌细胞一氧化氮合酶活性变化的异同，探讨一氧化氮合酶参与败血症休克性心肌抑制的机制。
Aim: To observe the differences of hemodynamics and nitric oxide synthase (NOS) activity of ventricular cardiac muscle in two septic shock models and explore the possible mechanism.
肠外抗生素预防联合抗生素骨水泥固定人工假体可有效预防败血症性休克，特别是在膝关节翻修术后。
The combination of parenteral antibiotic prophylaxis and prosthetic fixation with antibiotic-impregnated cement protected against septic failure, especially after revision knee arthroplasty.
单髁膝关节成形术较全髁膝关节成形术术后败血症性休克危险降低，但无显著性差别。
The rate of septic failure was lower after unicondylar than after total condylar primary knee arthroplasty, but the difference was not significant.
尿路败血症易并发休克，死亡率甚高。
Uric road septicemia is easy intercurrent shock, mortality is very tall.
败血症和感染性休克的危险因素包括：年龄大于60岁、急诊手术、存在合并症的患者。
Risk factors for sepsis and septic shock included age older than 60, the need for emergency surgery and the presence of any co-occurring illness.
持续性的菌血症经常导致严重的败血症，甚至恶化为败血性休克。
Persistent bacteremia often leads to severe sepsis or overwhelming septic shock.
院内感染、血小板下降、血糖升高及休克增加了败血症患者病死率。
The mortality rate associated with septicemia was related with the hospital onset of infection, decrease of platelet, elevation of blood glucose and septic shock.
例如，近年来科学家已经找到败血症引起发炎等一连串效应，终致休克的要素。
In recent years, for example, scientists have pinpointed elements of the inflammatory cascade triggered by sepsis, which ultimately leads to shock.
如不及时处理，败血症会恶化成败血性休克，其死亡率高达50%以上。
Without immediate treatment, septic shock follows, with a mortality rate over 50%.
败血症性休克常发生在细菌感染后，以机体免疫系统释放出大量的致炎(炎症前)细胞因子为特征。
Septic shock often follows a bacterial infection, and is characterized by the overwhelming release of pro-inflammatory cytokines by the body's immune system.
就败血症和感染性休克而言，无论是只清除某一种物质还是针对单个介导因子的干预手段似乎都不合适。
With regard to sepsis and septic shock, neither one-off interventions nor those directed at a single mediator appear appropriate.
由于细菌毒素的作用，尤其是内毒素，败血症性休克的促炎症性反应强于其他休克。
Septic shock may be more proinflammatory than other forms because of the actions of bacterial toxins, especially endotoxin.
任何一种休克后都可发生MODS，但以感染型最常见，器官衰竭是败血症性休克的典型特征之一。
MODS can follow any type of shock but is most common when infection is involved; organ failure is one of the defining features of septic shock.
任何一种休克后都可发生MODS，但以感染型最常见，器官衰竭是败血症性休克的典型特征之一。
MODS can follow any type of shock but is most common when infection is involved; organ failure is one of the defining features of septic shock.
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