TRAIL表达上调与雷帕霉素诱导血管内皮细胞损伤有一定的相关性。
The up-regulation of TRAIL might be related to the injury of vascular endothelial cells caused by rapamycin.
结果细胞因子、辐射、氧自由基等可诱导血管内皮细胞凋亡，导致血管损伤；
Results Cytokine, radiation could induce apoptosis of endothelial cells, and caused vascular damage.
实验证明，介导体可以通过诱导血管内皮细胞的编程性细胞死亡或吞噬来修复血管。
The work shows the intraceptor prompts regression of blood vessels by inducing programmed cell death, or apoptosis, in the vascular endothelial cells that line the vessels.
目的探讨血凝素样氧化低密度脂蛋白受体1 (LOX - 1)在氧化低密度脂蛋白(ox - LDL)诱导血管内皮细胞粘附分子表达中的作用。
Objective to investigate the effects of oxidized low-density lipoprotein receptor 1 (LOX-1) on secretion of adhesive molecules mediated by ox-LDL in human umbilical endothelial cells (HUVECs).
结论含高剂量中药的大鼠血清有抑制血管内皮细胞生长因子诱导内皮细胞增殖的作用。
CONCLUSION the rat serum including high dose herb medicine could have an effect on inhibition of proliferation of endothelial cell induced by vascular ECGF.
血管内皮生长因子（VEGF）是一类能促进血管生成的细胞因子，能诱导细胞的有丝分裂和调节内皮细胞的通透性。
The vascular endothelial growth factor (VEGF) is a kind of potent angiogenic cytokine that induces mitosis and regulates the permeability of endothelial cells.
目的：研究小鼠肺微血管内皮细胞分离培养方法并探讨脂多糖(LPS)诱导小鼠肺微血管内皮细胞血管内皮生长因子(VEGF)表达。
Objective: To establish a isolation and cultivation method of mice lung microvessel endothelial cell (LMVEC) and investigate the VEGF expression of mice LMVEC induced by lipopolysaccharide (LPS).
结论：脑缺血再灌诱导沙鼠脑微血管内皮细胞MMP - 9表达增加。
Conclusions: Increased MMP-9 expression is induced by cerebral ischemia and reperfusion of gerbil.
GM - CSF可以促进体外诱导人血管内皮细胞血管形成，并在一定范围内呈剂量及时间依赖效应。
GM-CSF promotes human vascular endothelial cells to form vessel-like structure in vitro in a dose-dependent and time-dependent manner.
结论：APC拮抗lps诱导的血管内皮细胞凋亡并促进细胞的增殖，发挥保护细胞的作用。
CONCLUSION: APC can inhibit HUVECs apoptosis induced by LPS and promote cell proliferation, thus protect the cells from injury.
结论牛磺酸对LDL诱导的血管内皮细胞损伤有保护作用，其保护作用与降低adma浓度有关。
Conclusion taurine protects the endothelium against damages induced by LDL, and the protective effect of taurine on endothelium is related to the reduction of ADMA concentration in rats.
结论麝香保心丸保护血管内皮细胞可能与抑制H2O2诱导人脐静脉内皮细胞氧化应激有关。
Conclusion SBP could protect endothelial cells and it might be related to inhibit oxidative stress induced by H2O2.
结论舒林酸的硫化代谢产物能够显著抑制血管内皮细胞的增殖活性，改变细胞的周期分布，使细胞阻滞于G 2期，并可能在此期诱导其凋亡。
Conclusions Sulfide can significantly reduce the proliferation of ECV304, change the cell cycle distribution and arrest cells in G2-M phase where apoptosis may be induced.
结论舒林酸的硫化代谢产物能够显著抑制血管内皮细胞的增殖活性，改变细胞的周期分布，使细胞阻滞于G 2期，并可能在此期诱导其凋亡。
Conclusions Sulfide can significantly reduce the proliferation of ECV304, change the cell cycle distribution and arrest cells in G2-M phase where apoptosis may be induced.
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