结论：APC拮抗lps诱导的血管内皮细胞凋亡并促进细胞的增殖，发挥保护细胞的作用。
CONCLUSION: APC can inhibit HUVECs apoptosis induced by LPS and promote cell proliferation, thus protect the cells from injury.
结论牛磺酸对LDL诱导的血管内皮细胞损伤有保护作用，其保护作用与降低adma浓度有关。
Conclusion taurine protects the endothelium against damages induced by LDL, and the protective effect of taurine on endothelium is related to the reduction of ADMA concentration in rats.
实验证明，介导体可以通过诱导血管内皮细胞的编程性细胞死亡或吞噬来修复血管。
The work shows the intraceptor prompts regression of blood vessels by inducing programmed cell death, or apoptosis, in the vascular endothelial cells that line the vessels.
结论含高剂量中药的大鼠血清有抑制血管内皮细胞生长因子诱导内皮细胞增殖的作用。
CONCLUSION the rat serum including high dose herb medicine could have an effect on inhibition of proliferation of endothelial cell induced by vascular ECGF.
目的探讨血凝素样氧化低密度脂蛋白受体1 (LOX - 1)在氧化低密度脂蛋白(ox - LDL)诱导血管内皮细胞粘附分子表达中的作用。
Objective to investigate the effects of oxidized low-density lipoprotein receptor 1 (LOX-1) on secretion of adhesive molecules mediated by ox-LDL in human umbilical endothelial cells (HUVECs).
血管内皮生长因子（VEGF）是一类能促进血管生成的细胞因子，能诱导细胞的有丝分裂和调节内皮细胞的通透性。
The vascular endothelial growth factor (VEGF) is a kind of potent angiogenic cytokine that induces mitosis and regulates the permeability of endothelial cells.
TRAIL表达上调与雷帕霉素诱导血管内皮细胞损伤有一定的相关性。
The up-regulation of TRAIL might be related to the injury of vascular endothelial cells caused by rapamycin.
结论舒林酸的硫化代谢产物能够显著抑制血管内皮细胞的增殖活性，改变细胞的周期分布，使细胞阻滞于G 2期，并可能在此期诱导其凋亡。
Conclusions Sulfide can significantly reduce the proliferation of ECV304, change the cell cycle distribution and arrest cells in G2-M phase where apoptosis may be induced.
结论舒林酸的硫化代谢产物能够显著抑制血管内皮细胞的增殖活性，改变细胞的周期分布，使细胞阻滞于G 2期，并可能在此期诱导其凋亡。
Conclusions Sulfide can significantly reduce the proliferation of ECV304, change the cell cycle distribution and arrest cells in G2-M phase where apoptosis may be induced.
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