目的研究丙泊酚对内毒素(LPS)诱导的大鼠急性肺损伤(ALI)的保护作用及其可能分子作用机制。
Objective To study the protective effect of propofol on acute lung injury (ALI) induced by lipopolysaccharide (LPS) in rats.
结论罗红霉素能改善大鼠急性肺损伤时气体交换能力，调节肺部细胞因子，对内毒素诱导的急性肺损伤有保护作用。
CONCLUSION Roxithromycin has a protective effect on rat acute lung injury induced by LPS through regulating cytokines, improving gas exchange ability, and inhibiting lung permeability.
结论增龄能够促进脂多糖诱导的急性肺损伤大鼠肺组织纤维蛋白沉积，增强纤维蛋白的内皮细胞损伤作用。
Conclusions Aging could accelerate fibrin deposition in pulmonary tissue of LPS-induced rat model of acute lung injury, and promote the injury of endothelial cell function caused by fibrin deposition.
目的探讨内毒素（LPS）诱导的急性肺损伤（ALI）大鼠肺组织甲状腺转录因子-1（TTF-1）、肺表面活性物质相关蛋白-A（SP-A）表达的变化。
Objective To investigate the change of the expression of TTF-1 and SP-A in lung tissue of ALI rats induced by LPS.
结论血必净可明显抑制脂多糖诱导的急性肺损伤大鼠肺组织增强的HMGB1表达，减轻脂多糖诱导的大鼠肺损伤程度。
Xuebijing could attenuate the degree of acute lung injury in rats induced by LPS, which may be related to its inhibition of the HMGB1 protein expression in lung tissue.
结论血必净可明显抑制脂多糖诱导的急性肺损伤大鼠肺组织增强的HMGB1表达，减轻脂多糖诱导的大鼠肺损伤程度。
Xuebijing could attenuate the degree of acute lung injury in rats induced by LPS, which may be related to its inhibition of the HMGB1 protein expression in lung tissue.
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