文章对脑缺血再灌注诱导内质网应激和upr的研究进展做了综述。
This article reviews the recent progress in er stress and UPR induced by cerebral ischemic-reperfusion.
目的：探讨内质网应激反应对于诱导病毒性心肌炎心肌细胞凋亡的作用。
Objective: to investigate the role of endoplasmic reticulum stress in induction of cardiac myocyte apoptosis in viral myocarditis.
结论：内质网应激反应可能对于诱导病毒性心肌炎的心肌细胞凋亡有一定作用。
Conclusion: These findings suggest endoplasmic reticulum stress may contribute to the cardiac myocyte apoptosis in viral myocarditis.
结论：内质网应激反应可能对于诱导病毒性心肌炎的心肌细胞凋亡有一定作用。
Conclusion: These findings suggest endoplasmic reticulum stress may contribute to the cardiac myocyte apoptosis in viral myocarditis.
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