利用D-半乳糖建立衰老模型。
三氯化铝所致急性衰老模型避暗法实验结果。
Results of escaping dark experiment of aluminium trichloride-induced model of senescence.
小鼠衰老模型采用D-半乳糖所致的亚急性衰老模型。
Subacute aging model which were caused by D-lactose was used to study.
研究黑果枸杞色素对半乳糖衰老模型小鼠抗衰老作用机理。
Senile mice models were established by galactose to study the anti-senile function and mechanism of Lycium Ruthenicum Murr. pigment.
目的：观察益寿调脂片对衰老模型小鼠自由基代谢调整作用。
To observe the effect of Yishou Tiaozhi tablet (YSTZT) on the metabolism of free radical in mice of aging model.
目的：考察西伯利亚白刺果油对衰老模型小鼠氧化能力的影响。
Objective: This paper is to study the effect of anti-oxidative in subacute aging model mice with Fruit oil of Nitraria sibirica Pall.
D半乳糖；摘除胸腺；神经细胞凋亡；线粒体膜电位；衰老模型。
Dgalactose; Thymus gland removal; Nerve cells apoptosis; Mitochondria membrane potential; Aging model.
方法体外培养人脐静脉内皮细胞，通过传代形成细胞的衰老模型。
Methods Human umbilical vein endothelial cells (HUVECs) were cultured in vitro.
目的：观察d -半乳糖衰老模型大鼠下丘脑弓状核超微结构的变化。
Objective: To observe the ultrastructural changes of arcuate nucleus in hypothalamus of D-gal aging rats.
目的：研究敦煌韦慈方对d半乳糖所致衰老模型小鼠的延缓衰老作用。
Objective: to study the effect of Dunhuang Weicifang delaying senility to D-galactose presenile model mice.
结论：以亚致死量H2O2处理年轻2BS细胞可以建立细胞衰老模型。
Conclusion:Senescent cell model could be established by 2BS treated with sub-lethal H2O2.
目的：研究敦煌大宝胶囊对衰老模型大鼠脑组织氧自由基域影响的影响。
Objective: To study effects of DunHuangShiShiDaBaoCapsule (DHDB) on oxygen free radical threshold in brain tissues of senile rats.
目的为探讨皮肤衰老机制及抗皮肤衰老药物研究提供较理想的皮肤衰老模型。
OBJECTIVE To establish a skin aging model for studying the process of skin aging and potential compounds against skin aging.
目的探讨平脉通颗粒对D -半乳糖所致亚急性衰老模型自由基代谢的影响。
Objective to investigate the effects of Pingmaitong granules on free radical metabolism of D-galactose-induced subacute aging model.
方法：采用小鼠注射d -半乳糖制成衰老模型。马齿苋水提液灌胃30d。
Methods: The senile mouse models induced by D-gal were prepared and given water extract of Machixian for 30 days.
方法：采用半乳糖腹腔注射制作大鼠衰老模型，分别用不同剂量的雷竹笋汁灌胃4周。
METHODS The mice with decrepit model induced by D-galactose (D-gal) were treated with LZS of different doses respectively for 4 weeks.
衰老模型是研究延缓衰老方法及其作用机制的关键一环，直接关系到实验结果的实际意义。
Aging model is a base to study the methods and the mechanism of delaying aging. The choice of what kind of aging model could directly affect the practical significance of experimental results.
研究结果提示：哈士蟆油及其有机相和无机相提取物均能够增强衰老模型小鼠学习记忆能力；
The result mentioned that the Oviductus Ranae, extracts of aqueous phase and benzene phase can improve aging model mice's learning & memory ability;
目的：探讨二至丸对亚急性衰老模型小鼠的抗氧化作用，为进一步研究其抗衰老作用提供依据。
Objective:To explore the antioxidation effects of Erzhiwan, for further research to provide the basis of its antiaging effects.
LBP和ABP能够改善H2O2诱导的衰老模型细胞的某些表型，这可能与其抗氧化作用有关。
LBP and ABP improved some phenotypes of cell model induced by H2O2, which might be related to their antioxidant effects.
结论DHDB可提高衰老模型动物脑组织单胺类神经递质的含量，对改善大脑功能有一定的作用。
Conclusions DHDB can upregulate monoamine neurotransmitter levels in the brain tissue of aging model rats and plays an important role in improving cerebral function.
本实验采用连续给予D-半乳糖的方法建立小鼠衰老模型，并观察中药“长寿灵”的抗衰老作用。
The aging model of mice made by D-galactose was employed in this experiment to study the antiaging effect of "Changshouling".
衰老模型实验：SD大鼠随机分为对照组、D-半乳糖组、正常用药组、溶媒对照组和药物治疗组。
SD rats were randomly assigned to five groups as follows: saline control group, D-galactose group, DHEA-treated normal group, vehicle control group and DHEA-treated senescent group.
目的通过d半乳糖诱导小鼠衰老模型，探讨缺锌对衰老小鼠抗氧化系统和肝脏dna损伤与修复的影响。
Objective to observe the effects of zinc-deficiency (ZD) on antioxidation system and repair of DNA damage and of hepatic cells by establishing an aging mice model induced with D-galactose.
目的：研究参芪王浆制剂抗自然衰老模型大鼠皮肤衰老的作用，并初步探讨该制剂延缓皮肤衰老的作用机理。
Objective: To discuss the skin anti-aging effect of Senqi Syrup on aged rats and explore the mechanism of action.
目的研究白果清蛋白(GAP)对自然衰老小鼠及半乳糖致亚急性衰老模型小鼠的延缓衰老作用，并探讨作用机制。
Aim By using natural aging mice and D-galactose-induced subacute aging mice, Anti-aging effects of GAP were studied and the mechanism of the effects was discussed.
作者在用抗体诱发动物急性溶血的方法建立的红细胞在体衰老模型的基础上，对红细胞衰老过程中的细胞密度变化进行了研究。
Changes in density of red blood cells (RBCs) during in vivo aging were studied using an animal model developed with the method of antibody inducement of acute RBC lysis.
由此提示，该红细胞衰老模型是可信的，能够用于研究红细胞的衰老过程并可以观察在衰老过程中与此相关的微观流变特性的改变。
Therefore, in can be implied that this RBC aging model is reliable, and it is favorable for the study of RBC aging process and the related microrheological changes in the time scale of days.
结果：健脑片能明显降低糖代谢衰老模型小鼠脑、肝匀浆及血浆中lpo水平，明显提高糖代谢衰老模型小鼠血液SOD、CAT活力。
Result: Jiannao tablet can obviously decrease the level of LOP in plasma, brain and liver homogenate, and increase the activity of SOD, CAT in blood of aging model mice.
结果：健脑片能明显降低糖代谢衰老模型小鼠脑、肝匀浆及血浆中lpo水平，明显提高糖代谢衰老模型小鼠血液SOD、CAT活力。
Result: Jiannao tablet can obviously decrease the level of LOP in plasma, brain and liver homogenate, and increase the activity of SOD, CAT in blood of aging model mice.
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