同时明显降低衰老小鼠脑组织MDA含量。
It could obviously reduce the content of MDA in the senile mice.
方法通过注射d半乳糖建立亚急性衰老小鼠模型。
Methods Subacute senile mouse model was established by injection of D-galactose.
方法通过注射D半乳糖建立亚急性衰老小鼠模型。
Methods Subacute senile mice model was established by injection of D-galactose.
目的：研究炒紫苏子醇提取物对衰老小鼠智力的影响。
Objective: To study the effect of roasted Perilla seed alcohol extract on the intelligence of ageing mice.
目的：观察银耳多糖对D -半乳糖所致衰老小鼠的作用。
Objective: to observe the protective effect of tremella polysaccharides (TP) on mouse aging model induced by D-galactose.
结论：二至丸能有效防止D -半乳糖致衰老小鼠的皮肤衰老。
Conclusion: Erzhi Pill can effectively resist the skin senility of the mouse with subacute senility resulting from D-galactose.
目的探讨CD 137分子在衰老小鼠脾脏T细胞表面表达的规律。
Aim To explore the CD137 expression on splenic t cells in senile mice.
研究山药水煎液对亚急性衰老小鼠游泳耐力及某些生理指标的影响。
Yam decoction can enhance the swimming endurance of the elder mice, protect the tissue stricture of immune organs and postpone the senescence process of mice to certain extent.
结论：芪附汤能明显地提高衰老小鼠的免疫功能，具有延缓衰老的作用。
CONCLUSIONS Qifu decoction could improve markedly the immune function of old age mice, and has anti-senility effect.
目的研究正常衰老小鼠小脑颗粒细胞膜膜电位及诱发动作电位的变化情况。
Objecetive To study the alterations in neuronal membrane potential (MP) and the induced action potential (AP) in cerebellar granule neurons in brain sections of aged mice.
结论：适宜游泳应激可改善衰老小鼠行为表现和免疫功能以延缓衰老进程。
Conclusion: Proper swim stress may delay aging process by improving the aging mice 'behavior and immune function.
目的：探讨葛根素对d -半乳糖致衰老小鼠学习记忆的影响及其作用机制。
AIM: to study the effects of puerarin on learning-memory behavior in aging mice induced by D-galactose and to explore in-brain mechanism of its effects.
目的：观察二至丸对D-半乳糖致亚急性衰老小鼠抗皮肤衰老的作用及机制。
Objective: To observe the actions and mechanism of Erzhi Pill for resisting the skin tissue senility of the mice with subacute senility resulting from D-galactose.
目的：探讨纳洛酮对三氯化铝所致急性衰老小鼠记忆障碍的保护作用及其机制。
OBJECTIVE: To investigate protective effects of naloxone on aluminium trichloride-induced memory impairment of senescence-accelerated mice and its mechanism.
结论火棘有一定的抗氧化作用，并能延缓由D -半乳糖所致衰老小鼠的衰老过程。
Conclusion It was suggested that Pyracanthy had some antioxidation and might prolong the aging process of mice model induced by overdose of D-galactose.
目的研究补肾益脑片对快速衰老小鼠（SAM鼠）生理及其脑组织基因表达的影响。
Objective To study the effects of Bushen Yin'ao Tablet (BSYNT) on physiology and cerebral gene expression in senescence-accelerated mice (SAM).
结论：龟鹿二仙胶颗粒对自然衰老小鼠具有增强抗氧化功能，提高学习记忆能力的作用。
Conclusion: the turtle and deer antlers granula can enhance antioxidant and improve learning and memory for the natural aging mice.
方法昆明种自然衰老小鼠45只随机分为老年空白对照组、老年阳性对照组、老年润肠丸组；
Methods 45 senile mice were randomly divided into senile blank control group, senile positive control group and senile Runchang pill group.
结论：肉苁蓉多糖具有明显的抗脂质过氧化功能，防止亚急性衰老小鼠的组织脂质过氧化损伤。
Conclusion:PCD exnibited significantly anti-lipid peroxide effect and prevented lipid peroxidative damages in tissues of the subacute aging mice.
目的通过d半乳糖诱导小鼠衰老模型，探讨缺锌对衰老小鼠抗氧化系统和肝脏dna损伤与修复的影响。
Objective to observe the effects of zinc-deficiency (ZD) on antioxidation system and repair of DNA damage and of hepatic cells by establishing an aging mice model induced with D-galactose.
表明OSAE可明显改善衰老小鼠学习记忆功能，提高抗氧化能力，从而可能具有较好的延缓衰老的作用。
It indicated that OSAE can improve the learning and memory ability and increase antioxidant capacity of aging mice, which may has a good anti-aging effect.
目的观察CD 137分子对衰老小鼠脾脏T细胞活化的影响，探讨该分子对衰老T细胞功能的调节作用。
Objective To observe the effect of CD137 on the activation of spleen t lymphocytes of senile mice and explore its regulative roles.
目的观察羧甲基壳聚糖（CMC）对黑腹果蝇寿命的影响及其对D-半乳糖致亚急性衰老小鼠的抗衰老作用。
Objective To observe the effect of carboxymethyl chitosan (CMC) on the lifespan of Drosophila melanogaster and D-galactose-induced aging in mice.
目的研究白果清蛋白(GAP)对自然衰老小鼠及半乳糖致亚急性衰老模型小鼠的延缓衰老作用，并探讨作用机制。
Aim By using natural aging mice and D-galactose-induced subacute aging mice, Anti-aging effects of GAP were studied and the mechanism of the effects was discussed.
结果表明TLG对自然衰老小鼠学习记忆障碍有一定的改善作用，其机制可能与增强机体抗氧化能力及抑制细胞凋亡有关。
The results showed TLG significantly improved learning and memory impairment of aging mice. It may be to enhance the ability of antioxidation and inhibit apoptosis of cerebral cell.
结果表明，健脑益肾冲剂能明显提高脑衰老小鼠学习记忆能力，降低其脑组织中LPO含量，并提高其脑组织中SOD活性。
The results show that it can clearly improve study memory ability of senile mice, and decrease LPO content and increase SOD activity in brain.
结论CT WA能拮抗自由基损伤，增强衰老小鼠心和脑组织端粒酶活性和机体免疫功能，这些可能与CT WA的抗衰老作用有关。
CONCLUSION CTWA may delay aging, which may be related to antagonizing free radical injury and enhancing immunity of aging mice.
水提液低、高剂量组能不同程度降低衰老小鼠脑、肝和血浆中MDA的含量，天冬总多糖高剂量组能降低衰老小鼠肝组织中MDA含量（P<0.01）。
The level of MDA in plasma, brain and liver of mice decreased in water soluble extracts, while the contents of MDA in the liver decreased in high dosage group of total polysaccharide(P<0.01).
水提液低、高剂量组能不同程度降低衰老小鼠脑、肝和血浆中MDA的含量，天冬总多糖高剂量组能降低衰老小鼠肝组织中MDA含量（P<0.01）。
The level of MDA in plasma, brain and liver of mice decreased in water soluble extracts, while the contents of MDA in the liver decreased in high dosage group of total polysaccharide(P<0.01).
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