补体激活造是炎症反应性疾病的病理反应之一。
Complement activation is the basic pathological reaction in inflammatory diseases.
胞外抑制素能够结合和激活C3从而增强补体激活(6)。
Extracellular prohibitin can bind and activate C3 to enhance complement activation (6).
眼镜蛇毒因子(CVF)是一种具有补体激活活性的糖蛋白。
Cobra venom factor (CVF) is a nontoxic glycoprotein present in cobra venom with strong complement-activating activity through the alternative pathway.
其在临床引起的不良反应可能为非补体激活相关的类过敏反应。
Therefore, the clinical adverse reactions of QKLI in clinic may be pseudoallergy which had no relation with the activation of complement system.
因此如何抑制补体激活，阻断其扩大的级联反应成为治疗急性胰腺炎的突破口。
So how to inhibit complement from activating and interrupt amplification of cascade reaction may be direction of treatment of AP.
除了在宿主防御中起到关键作用之外，补体激活还在许多病理情况下引起炎症反应和免疫应答。
Besides a critical role in innate host defence, complement activation contributes to inflammatory and immunological responses in a number of pathological conditions.
衰变加速因子（DAF）是补体激活途径中的一个重要膜调节蛋白，转人DAF基因猪可以作为人器官移植的供体。
Decay accelerating factor(DAF)is one of the important membrane regulating protein in the complement activating ways. The transgenic pigs for human DAF gene can used for xenotransplatations donors.
本文就褐藻糖胶的抗血栓、抗肿瘤、抗氧化、调节血脂、抗病毒、抗辐射和抑制补体激活等药理作用及其作用机制进行综述。
This review summarized its pharmacological activities and related mechanism such as antithrombotic, antitumor, antioxidation, hypolipide, antivirus, antiradiation and anti-activation of complement.
由于每个抗体分子可以激活几百个补体分子，这样可以使抗原抗体反应得到充分的作用。
Because each immune body member may activate several hundred complement members, like this may cause the antigen immune body to respond that obtains the full function.
结果陈株、H8205、R 22 3株病毒抗原，可以直接激活补体C_3，电泳后呈现典型降解峰，而ALC96与正常细胞抗原则均为阴性。
The results showed that Chen, H8205, R22 strains antigens can directly activate C_3, typical degradative peak was found after electrophoresis. But ALC96 and normal antigen were negative.
HE膜用过氧乙酸和福尔·马林消毒复用可在一定程度降低其激活补体的能力。
Reused HE, which sterilized with peroxide or formalin, decreases its ability of complement activation.
方法采用眼镜蛇毒因子特异激活血清补体，诱导人神经母细胞瘤细胞SH-SY5Y损伤。
Methods A model of neuronal injury was established on SH-SY5Y cells through activation of complement by cobra venom factor, an alternative pathway-specific activator.
脑出血时脑内补体系统激活，并通过多种机制导致组织损伤。
The complement cascade is activated during ICH, and it might cause tissue damage through variety of mechanisms.
结果LPS能够激活血清补体，这种激活呈现量效、时效性。
Results LPS activated complement in a dose-and time-dependent manner.
结果LPS能够激活血清补体，这种激活呈现量效、时效性。
Results LPS activated complement in a dose-and time-dependent manner.
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