血纤维蛋白原和凝血酶，自身手创伤时使血液凝固以堵塞血管破口，防止血液流失过多。

Fibrinogen and thrombin may concrete blood to jam the crevasses of blood vessels for avoiding excessive loss of blood.

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同样地，高磷酸盐血症、酸液过多症、蛋白尿的存在，依靠试验性模型和饮食方式，可能使人误解，并且无法对其扩展性表现出的准确测量。

Likewise, the presence of hyperphosphatemia, acidosis, and proteinuria may be misleading depending on the experimental model and diet and may not represent a precise measure of progression.

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过量的蛋白质供给可导致血氨过高或血尿素氮升高，进而可发生液体负荷过多，胆汁淤积和渗透性利尿。

Excess protein may cause hyperammonemia or an elevated BUN. Fluid overload, cholestasis and osmotic diuresis are further problems which may occur.

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例如，家族性血胆脂醇过多症(FH)通常起源于编码低密度脂蛋白(LDL)受体的LDLR突变。

For instance, familial hypercholesterolemia (FH) typically results from mutations in LDLR encoding the low-density lipoprotein (LDL) receptor.

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例如，家族性血胆脂醇过多症(FH)通常起源于编码低密度脂蛋白(LDL)受体的LDLR突变。

For instance, familial hypercholesterolemia (FH) typically results from mutations in LDLR encoding the low-density lipoprotein (LDL) receptor.

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