血纤维蛋白原和凝血酶，自身手创伤时使血液凝固以堵塞血管破口，防止血液流失过多。
Fibrinogen and thrombin may concrete blood to jam the crevasses of blood vessels for avoiding excessive loss of blood.
目的对一个遗传性异常纤维蛋白原血症家系进行表型和基因型分析。
Objective To analyze the phenotype and genotype of a family with inherited dysfibrinogenemia.
遗传性无纤维蛋白原血症是一种由于纤维蛋白原基因缺陷所致常染色体隐性遗传病。
Congenital afibrinogenemia is a rare autosomal recessive disorder, characterized by the complete absence or extremely reduced level of fibrinogen.
结论纤维蛋白原FGA基因复合杂合缺失是引起该家系先证者无纤维蛋白原血症的原因。
Conclusion Inherited afibrinogenemia is caused by the compound heterozygous deletion in the fibrinogen FGA gene.
结论：降纤酶治疗急性脑梗死疗效确切，能显著降低纤维蛋白原，改善全血黏度，降低致残率，不良反应少。
Conclusion: Defibrase can significantly decrease fibrinogen, improve whole blood viscosity, and decrease disability rate. With little side effect, it is effective on acute cerebral infarction.
两组于治疗前、后分别采静脉血检测血浆纤维蛋白原、红细胞压积、全血粘度和血浆粘度的变化。
Venous blood samples were taken respectively in the two groups to detect the changes of plasma fibrinogen, hematocrit, whole blood viscosity and plasma viscosity.
还分别测定了全血黏度、浆黏度、细胞聚集指数(IRC)纤维蛋白原及血小板聚集率。
Blood viscosity, plasma viscosity, aggregation index of RBC (AIRC) and fibrinogen were also assayed respectively.
降纤酶有明显降低血黏度、纤维蛋白原含量、红细胞聚集性、血小板聚集作用。
Defibrase can effectively reduce blood viscosity, content of fibrinogen, hemocyte aggregation and platelet aggregation.
结论：高血压、血纤溶系统活性升高、高水平纤维蛋白原、高同型半胱氨酸血症是脑梗死再发的重要危险因素。
Conclusion Hypertension, increased plasma PAI-1 activity, fibrinogen level and plasma Hcy level are the important risk factors in the recurrent cerebral infarction.
结果脂肪肝患者的全血高切黏度、中切黏度、低切黏度、血浆黏度、红细胞聚集指数和纤维蛋白原均高于非脂肪肝人员（P〈0.01）。
Results All the values of whole blood viscosity, plasma viscosity, red cell aggregation index and plasma fibrinogen of the patients with fatty liver were higher than those of healthy people(P0.01).
方法对T IA患者和健康对照组各50例进行整夜多导睡眠检测，以及血常规、血脂、血黏度、纤维蛋白原检测。
Methods: 50 patients with TIA and 50 controls from the health were performed overnight polysomnography as well as blood routine, lipidemia, blood viscosity and fibrinogen.
结果：发现ION组全血黏度、血浆度、红细胞电泳时间及血浆纤维蛋白原含量增高，与对照组差别显著（P< 0 .0 5 ，P< 0 .0 1 ）。
Results: There was significant differences in blood viscosity, plasma viscosity, erythrocyte electrophoresis time and fibrinogen level between ION group and matched control (P<0.05, P<0.01).
结果：发现ION组全血黏度、血浆度、红细胞电泳时间及血浆纤维蛋白原含量增高，与对照组差别显著（P< 0 .0 5 ，P< 0 .0 1 ）。
Results: There was significant differences in blood viscosity, plasma viscosity, erythrocyte electrophoresis time and fibrinogen level between ION group and matched control (P<0.05, P<0.01).
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