过大的氧化压力可能使磷酸己糖支路或高铁血红蛋白还原酶途径不堪重负，分别造成海因茨小体性溶血或形成高铁血红蛋白。
Excessive oxidant stress may overload the protective HMP shunt or methemoglobin reductase pathways, causing Heinz body hemolysis or methemoglobin formation, respectively.
当发生血管内溶血性贫血，更多红细胞在血液循环(血红蛋白血症)被破坏以至于超出肝球蛋白质的结合能力。
In intravascular hemolytic anemia, more RBC are destroyed in the circulation (hemoglobinemia) than can be bound to haptoglobin.
溶血介绍了分手的红血细胞，导致在释放血红蛋白进入血浆。
Haemolysis describes the breaking up of red blood cells, resulting in the release of haemoglobin into the plasma.
不过，现在这些过多的游离血红蛋白能在血浆中被检测出，因此，本文调查研究了植入不同类型VAD的患者出现溶血的情况。
Only now, the excessive free haemoglobin can be measured in plasma. Here, we investigated haemolysis in patients supported by different VAD types.
目的观察平均红细胞血红蛋白浓度（MCHC）检测对溶血性贫血（HA）的诊断价值。
Objective The study was conducted to evaluate the diagnostic value of the mean corpuscular hemoglobin concentration(MCHC) in hemolytic anemia(HA).
所有患者都具有急性溶血性贫血，黄疸等临床表现及不同程度的血红蛋白尿、G6PD酶活性降低。
All these cases had acute hemolytic anemia, jaundice, hemoglobinuria and reduced G6PD activity to different extents.
大负荷的运动训练导致溶血发生，使运动员出现运动性低血红蛋白甚至贫血现象，严重影响运动成绩和身体健康。
Hemolysis may happen during the process of the heavy-load exercise, which can result in sports hypochromia or even anemia, and badly affect athletes' sports level and their health.
结论G 6 PD溶血虽然是自限性的，但当合并地中海贫血时，往往加重病情，尤其是某些特殊类型的异常血红蛋白。
ConclusionG6PD hemolysis is a self-limited disease, but it can grew worse when it is complicated by thalassemias, especially with some special types of abnormal hemoglobin.
结论G 6 PD溶血虽然是自限性的，但当合并地中海贫血时，往往加重病情，尤其是某些特殊类型的异常血红蛋白。
ConclusionG6PD hemolysis is a self-limited disease, but it can grew worse when it is complicated by thalassemias, especially with some special types of abnormal hemoglobin.
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